G. Hotamisligil

A central role for JNK in obesity and insulin resistance

Michael Karin, M. Karin, Lufen Chang, 2002, Nature.

Adipose Tissue Resistin Expression Is Severely Suppressed in Obesity and Stimulated by Peroxisome Proliferator-activated Receptor γ Agonists*

T. Willson, S. Kliewer, K. Brown, 2001, The Journal of Biological Chemistry.

Protection from obesity-induced insulin resistance in mice lacking TNF-α function

K. Uysal, S. Wiesbrock, M. Marino, 1997, Nature.

Tumor necrosis factor alpha mediates apoptosis of brown adipocytes and defective brown adipocyte function in obesity.

E. Nisoli, K. Uysal, S. Wiesbrock, 2000, Proceedings of the National Academy of Sciences of the United States of America.

Tumor necrosis factor-alpha contributes to obesity-related hyperleptinemia by regulating leptin release from adipocytes.

T. Kirchgessner, K. Uysal, S. Wiesbrock, 1997, The Journal of clinical investigation.

Altered insulin secretion associated with reduced lipolytic efficiency in aP2-/- mice.

R. Parker, K. Uysal, S. Wiesbrock, 1999, Diabetes.

JNK1 deficiency does not enhance muscle glucose metabolism in lean mice.

N. Fujii, L. Goodyear, J. Brandauer, 2006, Biochemical and biophysical research communications.

Upregulation of uncoupling protein 2 mRNA in genetic obesity: lack of an essential role for leptin, hyperphagia, increased tissue lipid content, and TNF-alpha.

R. Faggioni, K. Feingold, C. Grunfeld, 2000, Biochimica et biophysica acta.