Q. Zheng-Fischhöfer

发表

Protein kinase MARK/PAR-1 is required for neurite outgrowth and establishment of neuronal polarity.

E. Mandelkow, E. Mandelkow, Yong-zhong Wu, 2002, Molecular biology of the cell.

Structure, microtubule interactions, and paired helical filament aggregation by tau mutants of frontotemporal dementias.

E. Mandelkow, Q. Zheng-Fischhöfer, J. Biernat, 2000, Biochemistry.

The endogenous and cell cycle-dependent phosphorylation of tau protein in living cells: implications for Alzheimer's disease.

E. Mandelkow, E. Mandelkow, U. Preuss, 1998, Molecular biology of the cell.

Characterization of connexin30.3-deficient mice suggests a possible role of connexin30.3 in olfaction.

J. Degen, K. Willecke, E. Dere, 2007, European journal of cell biology.

Connexin31 cannot functionally replace connexin43 during cardiac morphogenesis in mice

K. Willecke, A. Ghanem, J. Nagy, 2006, Journal of Cell Science.

Characterization of connexin31.1-deficient mice reveals impaired placental development.

K. Willecke, M. Kretz, S. Lye, 2007, Developmental biology.

Connexin31.1 deficiency in the mouse impairs object memory and modulates open-field exploration, acetylcholine esterase levels in the striatum, and cAMP response element-binding protein levels in the striatum and piriform cortex

K. Willecke, E. Dere, J. Huston, 2008, Neuroscience.

Sequential phosphorylation of Tau by glycogen synthase kinase-3beta and protein kinase A at Thr212 and Ser214 generates the Alzheimer-specific epitope of antibody AT100 and requires a paired-helical-filament-like conformation.

E. Mandelkow, E. Mandelkow, S. Illenberger, 1998, European journal of biochemistry.

Connexin 30.3 Is Expressed in the Kidney But Not Regulated by Dietary Salt or High Blood Pressure

K. Willecke, J. Peti-Peterdi, A. McDonough, 2008, Cell Communication & Adhesion.