F. C. de Beer

发表

Serum amyloid A protein and C-reactive protein levels in pulmonary tuberculosis: relationship to amyloidosis.

R. Gie, F. D. de Beer, A. Nel, 1984, Thorax.

High-Density Lipoprotein Suppresses the Type I Interferon Response, a Family of Potent Antiviral Immunoregulators, in Macrophages Challenged With Lipopolysaccharide

Roger E Bumgarner, Masashi Suzuki, R. Beyer, 2010, Circulation.

Increased serum amyloid a levels reflect colitis severity and precede amyloid formation in IL-2 knockout mice.

M. Kindy, F. D. de Beer, W. D. de Villiers, 2000, Cytokine.

Alternative forms of the scavenger receptor BI (SR-BI).

P. Connell, N. Webb, F. D. de Beer, 1997, Journal of lipid research.

Hepatocytes direct the formation of a pro-metastatic niche in the liver.

M. Borad, Dexi Liu, S. Yee, 2019, Nature.

C-reactive protein in SLE.

M. Pepys, F. D. de Beer, J. Lanham, 1982, Clinics in rheumatic diseases.

Binding of serum amyloid P-component (SAP) by amyloid fibrils.

M. Skinner, M. Pepys, F. D. de Beer, 1979, Clinical and experimental immunology.

Structure, organization, and chromosomal mapping of the gene encoding macrosialin, a macrophage-restricted protein.

F. D. de Beer, W. D. de Villiers, D. R. van der Westhuyzen, 1998, Genomics.

Serum Amyloid A Is an Exchangeable Apolipoprotein

P. McNamara, P. Shridas, N. Webb, 2018, Arteriosclerosis, thrombosis, and vascular biology.

Serum amyloid A impairs the antiinflammatory properties of HDL.

S. Subramanian, A. Chait, K. Elkon, 2016, The Journal of clinical investigation.

Deficiency of Endogenous Acute-Phase Serum Amyloid A Protects apoE−/− Mice From Angiotensin II–Induced Abdominal Aortic Aneurysm Formation

R. Charnigo, A. Daugherty, D. Rateri, 2015, Arteriosclerosis, thrombosis, and vascular biology.

Serum amyloid A, but not C-reactive protein, stimulates vascular proteoglycan synthesis in a pro-atherogenic manner.

V. King, N. Webb, F. D. de Beer, 2008, The American journal of pathology.

Serum amyloid A protein family. Differential induction by oxidized lipids in mouse strains.

A. Lusis, J. Berliner, M. Kindy, 1994, Arteriosclerosis and thrombosis : a journal of vascular biology.

Serum amyloid A-containing human high density lipoprotein 3. Density, size, and apolipoprotein composition.

G. Coetzee, F. D. de Beer, A. Strachan, 1986, The Journal of biological chemistry.

SR-BII, an Isoform of the Scavenger Receptor BI Containing an Alternate Cytoplasmic Tail, Mediates Lipid Transfer between High Density Lipoprotein and Cells*

P. Connell, N. Webb, G. Graf, 1998, The Journal of Biological Chemistry.

Low-Density Lipoprotein From Apolipoprotein E–Deficient Mice Induces Macrophage Lipid Accumulation in a CD36 and Scavenger Receptor Class A–Dependent Manner

F. D. de Beer, W. D. de Villiers, D. R. van der Westhuyzen, 2004, Arteriosclerosis, thrombosis, and vascular biology.

CD36 does not play a direct role in HDL or LDL metabolism.

N. Webb, F. D. de Beer, W. D. de Villiers, 2001, Journal of lipid research.

Remodeling of HDL remnants generated by scavenger receptor class B type I Published, JLR Papers in Press, June 21, 2004. DOI 10.1194/jlr.M400026-JLR200

N. Webb, F. D. de Beer, B. Asztalos, 2004, Journal of Lipid Research.

The fate of HDL particles in vivo after SR-BI-mediated selective lipid uptake Published, JLR Papers in Press, August 16, 2002. DOI 10.1194/jlr.M200173-JLR200

M. Kindy, N. Webb, F. D. de Beer, 2002, Journal of Lipid Research.

Expression of mouse acute-phase (SAA1.1) and constitutive (SAA4) serum amyloid A isotypes: influence on lipoprotein profiles.

M. Kindy, F. D. de Beer, J. Yu, 2000, Arteriosclerosis, thrombosis, and vascular biology.

Inflammation-Dependent Cerebral Deposition of Serum Amyloid A Protein in a Mouse Model of Amyloidosis

Jun-tao Guo, M. Kindy, F. D. de Beer, 2002, The Journal of Neuroscience.

Group X secretory phospholipase A(2) augments angiotensin II-induced inflammatory responses and abdominal aortic aneurysm formation in apoE-deficient mice.

M. Gelb, A. Daugherty, Deborah A. Howatt, 2011, Atherosclerosis.

SAA does not induce cytokine production in physiological conditions.

N. Webb, F. D. de Beer, Myung-hee Kim, 2013, Cytokine.

The Impairment of Macrophage-to-Feces Reverse Cholesterol Transport during Inflammation Does Not Depend on Serum Amyloid A

Jason M. Meyer, N. Webb, F. D. de Beer, 2013, Journal of lipids.

SR-BI protects against endotoxemia in mice through its roles in glucocorticoid production and hepatic clearance.

F. D. de Beer, D. R. van der Westhuyzen, A. Ji, 2008, The Journal of clinical investigation.

Intestinal Epithelial Serum Amyloid A Modulates Bacterial Growth In Vitro and Pro-Inflammatory Responses in Mouse Experimental Colitis

R. Arsenescu, F. D. de Beer, W. D. de Villiers, 2010, BMC gastroenterology.

C-reactive protein and apoB containing lipoproteins are associated with Mycobacterium leprae in lesions of human leprosy.

D. Ridley, M. Pepys, F. D. de Beer, 1984, Clinical and experimental immunology.

Rabbit and rat C-reactive proteins bind apolipoprotein B-containing lipoproteins

A. Feinstein, M. Pepys, A. Soutar, 1984, The Journal of experimental medicine.

Low density lipoprotein and very low density lipoprotein are selectively bound by aggregated C-reactive protein

A. Feinstein, M. Pepys, A. Soutar, 1982, The Journal of experimental medicine.

PHYLOGENETIC ASPECTS OF C‐REACTIVE PROTEIN AND RELATED PROTEINS *

A. Feinstein, F. D. de Beer, J. Clamp, 1982, Annals of the New York Academy of Sciences.

Isolation of human C-reactive protein and serum amyloid P component.

M. Pepys, F. D. de Beer, F. C. de Beer, 1982, Journal of immunological methods.

SERUM AMYLOID-A PROTEIN CONCENTRATION IN INFLAMMATORY DISEASES AND ITS RELATIONSHIP TO THE INCIDENCE OF REACTIVE SYSTEMIC AMYLOIDOSIS

M. Pepys, F. Beer, E. Fagan, 1982, The Lancet.

Characterization of constitutive human serum amyloid A protein (SAA4) as an apolipoprotein.

M. Kindy, F. D. de Beer, B. Asztalos, 1995, Journal of Lipid Research.

HDL Remodeling During the Acute Phase Response

N. Webb, F. D. de Beer, D. R. van der Westhuyzen, 2009, Arteriosclerosis, thrombosis, and vascular biology.

Influence of apoA-I and apoE on the formation of serum amyloid A-containing lipoproteins in vivo and in vitro Published, JLR Papers in Press, November 1, 2003. DOI 10.1194/jlr.M300414-JLR200

G. Getz, N. Webb, F. D. de Beer, 2004, Journal of Lipid Research.

HDL modification by secretory phospholipase A(2) promotes scavenger receptor class B type I interaction and accelerates HDL catabolism.

P. Connell, N. Webb, F. D. de Beer, 2000, Journal of lipid research.

Serum amyloid A impairs the antiinflammatory properties of HDL.

S. Subramanian, A. Chait, K. Elkon, 2016, The Journal of clinical investigation.

Human serum amyloid A protein. Behaviour in aqueous and urea-containing solutions and antibody production.

G. Coetzee, F. D. de Beer, A. Strachan, 1989, The Biochemical journal.

Structural prerequisites for serum amyloid A fibril formation.

C. Kay, M. Kindy, F. D. de Beer, 1993, The Journal of biological chemistry.

Mouse serum amyloid A protein. Complete amino acid sequence and mRNA analysis of a new isoform.

J. Sipe, F. D. de Beer, I. Carreras, 1992, The Biochemical journal.

Serum Amyloid A Promotes Inflammation-Associated Damage and Tumorigenesis in a Mouse Model of Colitis-Associated Cancer

A. Engelbrecht, P. Shridas, F. D. de Beer, 2021, Cellular and molecular gastroenterology and hepatology.

Correlation in rheumatoid arthritis of concentrations of plasma C3d, serum rheumatoid factor, immune complexes and C-reactive protein with each other and with clinical features of disease activity.

D. Vergani, M. Pepys, F. D. de Beer, 1982, Clinical and experimental immunology.

Adipocyte-Derived Serum Amyloid A Promotes Angiotensin II–Induced Abdominal Aortic Aneurysms in Obese C57BL/6J Mice

L. Cassis, P. Shridas, N. Webb, 2022, Arteriosclerosis, thrombosis, and vascular biology.

A Murine Model of Obesity With Accelerated Atherosclerosis

V. King, F. D. de Beer, Lisa R Tannock, 2010, Obesity.

Measurement of serum C-reactive protein concentration in myocardial ischaemia and infarction.

A. Maseri, M. Pepys, K. Fox, 1982, British heart journal.

Serum Amyloid A Promotes Cholesterol Efflux Mediated by Scavenger Receptor B-I*

F. D. de Beer, D. R. van der Westhuyzen, L. Cai, 2005, Journal of Biological Chemistry.

Serum Amyloid A Is a Ligand for Scavenger Receptor Class B Type I and Inhibits High Density Lipoprotein Binding and Selective Lipid Uptake*

F. D. de Beer, D. R. van der Westhuyzen, L. Cai, 2005, Journal of Biological Chemistry.

Apolipoprotein A-II Modulates the Binding and Selective Lipid Uptake of Reconstituted High Density Lipoprotein by Scavenger Receptor BI*

A. Jonas, N. Webb, F. D. de Beer, 2001, The Journal of Biological Chemistry.

Identification of novel members of the serum amyloid A protein superfamily as constitutive apolipoproteins of high density lipoprotein.

William Arbuthnot Sir Lane, J. Lélias, A. Whitehead, 1992, The Journal of biological chemistry.

Group V Secretory Phospholipase A2 Promotes Atherosclerosis: Evidence From Genetically Altered Mice

C. Jordan, M. Wadsworth, D. Taatjes, 2007, Arteriosclerosis, thrombosis, and vascular biology.

SR-BI-mediated selective lipid uptake segregates apoA-I and apoA-II catabolism Published, JLR Papers in Press, August 1, 2005. DOI 10.1194/jlr.M500068-JLR200

N. Webb, F. D. de Beer, D. R. van der Westhuyzen, 2005, Journal of Lipid Research.

Overexpression of SR-BI by adenoviral vector promotes clearance of apoA-I, but not apoB, in human apoB transgenic mice DOI 10.1194/jlr.M200026-JLR200

A. Daugherty, M. Kindy, N. Webb, 2002, Journal of Lipid Research.

Adenoviral vector-mediated overexpression of serum amyloid A in apoA-I-deficient mice.

M. Kindy, N. Webb, F. D. de Beer, 1997, Journal of lipid research.

SR-BI Selective Lipid Uptake: Subsequent Metabolism of Acute Phase HDL

N. Webb, F. D. de Beer, D. R. van der Westhuyzen, 2009, Arteriosclerosis, thrombosis, and vascular biology.

ApoA-II modulates the association of HDL with class B scavenger receptors SR-BI and CD36 Published, JLR Papers in Press, January 16, 2004. DOI 10.1194/jlr.M300417-JLR200

A. Stromberg, L. Castellani, F. D. de Beer, 2004, Journal of Lipid Research.

Functional effects of CRP binding to nuclei.

P. V. van Helden, F. D. de Beer, E. Shephard, 1986, Immunology.

CRP and neutrophils: functional effects and complex uptake.

R. Anderson, A. Strachan, E. Shephard, 1986, Clinical and Experimental Immunology.

Neutrophil lysosomal degradation of human CRP: CRP-derived peptides modulate neutrophil function.

R. Anderson, F. D. de Beer, E. Shephard, 1988, Clinical and experimental immunology.

Neutrophil association and degradation of normal and acute-phase high-density lipoprotein 3.

G. Coetzee, F. D. de Beer, D. R. van der Westhuyzen, 1987, The Biochemical journal.

Serum amyloid A3 is a high density lipoprotein-associated acute-phase protein

A. Chait, P. Shridas, N. Webb, 2017, Journal of Lipid Research.

Serum Amyloid A Facilitates the Binding of High-Density Lipoprotein From Mice Injected With Lipopolysaccharide to Vascular Proteoglycans

A. Chait, T. Vaisar, J. Heinecke, 2011, Arteriosclerosis, thrombosis, and vascular biology.

ApoB-containing lipoproteins in apoE-deficient mice are not metabolized by the class B scavenger receptor BI Published, JLR Papers in Press, October 16, 2003. DOI 10.1194/jlr.M300319-JLR200

N. Webb, F. D. de Beer, D. R. van der Westhuyzen, 2004, Journal of Lipid Research.

Overexpression of Secretory Phospholipase A2 Causes Rapid Catabolism and Altered Tissue Uptake of High Density Lipoprotein Cholesteryl Ester and Apolipoprotein A-I*

D. Rader, U. Tietge, F. D. de Beer, 2000, The Journal of Biological Chemistry.

Expression of serum amyloid A protein in the absence of the acute phase response does not reduce HDL cholesterol or apoA-I levels in human apoA-I transgenic mice.

D. Rader, M. S. Purdom, U. Tietge, 1999, Journal of lipid research.

Serum amyloid A protein enhances the activity of secretory non-pancreatic phospholipase A2.

P. Vadas, W. Pruzanski, F. D. de Beer, 1995, The Biochemical journal.

Calcium-dependent aggregation of human serum amyloid P component.

A. Feinstein, M. Pepys, F. D. de Beer, 1982, Biochimica et biophysica acta.

HDL cholesterol transport during inflammation

N. Webb, F. D. de Beer, D. R. van der Westhuyzen, 2007, Current opinion in lipidology.

Serum amyloid A augments the atherogenic effects of cholesteryl ester transfer protein

P. Shridas, N. Webb, F. D. de Beer, 2023, Journal of lipid research.

ATP binding cassette G1-dependent cholesterol efflux during inflammation1

A. Vaughan, N. Webb, F. D. de Beer, 2011, Journal of Lipid Research.

Macrophage-Expressed Group IIA Secretory Phospholipase A2 Increases Atherosclerotic Lesion Formation in LDL Receptor-Deficient Mice

A. Daugherty, N. Webb, Meredith A. Bostrom, 2003, Arteriosclerosis, thrombosis, and vascular biology.

Cerebrospinal fluid C-reactive protein in infective meningitis in childhood.

F. D. de Beer, P. Donald, A. Strachan, 1985, The Journal of laboratory and clinical medicine.

Impact of individual acute phase serum amyloid A isoforms on HDL metabolism in mice[S]

R. Charnigo, N. Webb, D. R. van der Westhuyzen, 2016, Journal of Lipid Research.

Solid-phase immunoradiometric assay for serum amyloid A protein using magnetisable cellulose particles.

M. Pepys, F. D. de Beer, R. Dyck, 1982, Journal of immunological methods.

Impact of serum amyloid A on high density lipoprotein composition and levels

D. Rateri, N. Webb, D. R. van der Westhuyzen, 2010, Journal of Lipid Research.

Identification of a novel serum amyloid A protein in BALB/c mice.

S. Shedlofsky, F. D. de Beer, M. de Beer, 1991, The Biochemical journal.

Antisense Oligonucleotide Targeting Hepatic Serum Amyloid A Limits the Progression of Angiotensin II-Induced Abdominal Aortic Aneurysm Formation

P. Shridas, N. Webb, A. Ji, 2023, bioRxiv.

Adipocyte-Derived Serum Amyloid A Promotes Angiotensin II–Induced Abdominal Aortic Aneurysms in Obese C57BL/6J Mice

L. Cassis, P. Shridas, N. Webb, 2022, Arteriosclerosis, thrombosis, and vascular biology.

Deficiency of Endogenous Acute Phase Serum Amyloid A Does Not Affect Atherosclerotic Lesions in Apolipoprotein E–Deficient Mice

A. Daugherty, D. Rateri, Deborah A. Howatt, 2014, Arteriosclerosis, thrombosis, and vascular biology.

Association of apolipoprotein E with murine amyloid A protein amyloid.

M. Kindy, F. D. de Beer, M. de Beer, 1995, Laboratory investigation; a journal of technical methods and pathology.

Serum Amyloid A is not obligatory for high-fat, high-sucrose, cholesterol-fed diet-induced obesity and its metabolic and inflammatory complications

P. Shridas, N. Webb, F. D. de Beer, 2022, PloS one.

Human serum amyloid A protein. Complete amino acid sequence of a new variant.

J. Sipe, F. D. de Beer, M. de Beer, 1992, The Biochemical journal.

Human serum amyloid A protein. The assignment of the six major isoforms to three published gene sequences and evidence for two genetic loci.

G. Coetzee, F. D. de Beer, A. Strachan, 1989, The Journal of biological chemistry.

Identification of three isoform patterns of human serum amyloid A protein.

G. Coetzee, F. D. de Beer, A. Strachan, 1988, The Biochemical journal.

Acute phase response in bronchiectasis and bronchus carcinoma.

F. D. de Beer, A. Nel, A. Strachan, 1984, Respiration; international review of thoracic diseases.

Deficiency of Acute-Phase Serum Amyloid A Exacerbates Sepsis-Induced Mortality and Lung Injury in Mice

P. Shridas, N. Webb, Lisa R Tannock, 2023, International journal of molecular sciences.

Nascent HDL formation by hepatocytes is reduced by the concerted action of serum amyloid A and endothelial lipase

N. Webb, F. D. de Beer, D. R. van der Westhuyzen, 2011, Journal of Lipid Research.