Y. Kaneda

Inhibition of TGF-beta 1 expression by antisense oligonucleotides suppressed extracellular matrix accumulation in experimental glomerulonephritis.

Y. Kaneda, E. Imai, Y. Akagi, 1996, Kidney international.

In vivo transfection of genes for renin and angiotensinogen into the glomerular cells induced phenotypic change of the mesangial cells and glomerular sclerosis.

Y. Kaneda, E. Imai, Y. Akagi, 1995, Biochemical and biophysical research communications.

Glomerulosclerosis induced by in vivo transfection of transforming growth factor-beta or platelet-derived growth factor gene into the rat kidney.

Y. Kaneda, Y. Isaka, E. Imai, 1993, The Journal of clinical investigation.

Gene transfer into the glomerulus by the hemagglutinating virus of Japan-liposome method.

Y. Akagi, Y. Isaka, E. Imai, 1997, Experimental nephrology.

Hepatocyte growth factor gene therapy reduces ventricular arrhythmia in animal models of myocardial ischemia . ∗

R. Morishita, Motokuni Aoki, T. Ohe, 2010 .

Transfection of rat kidney with human 15-lipoxygenase suppresses inflammation and preserves function in experimental glomerulonephritis.

Y. Kaneda, K. Badr, E. Imai, 1999, Proceedings of the National Academy of Sciences of the United States of America.

Gene therapy by transforming growth factor-beta receptor-IgG Fc chimera suppressed extracellular matrix accumulation in experimental glomerulonephritis.

Y. Kaneda, M. Hori, E. Imai, 1999, Kidney international.

Transforming growth factor-beta 1 antisense oligodeoxynucleotides block interstitial fibrosis in unilateral ureteral obstruction.

E. Imai, Hiroyuki Nakamura, Y. Ando, 2000, Kidney international.

Introduction of a foreign gene into the kidney in vivo: development of glomerulosclerosis by the transfection of genes for PDGF and TGF-beta.

Y. Kaneda, E. Imai, Y. Isaka, 1994, Contributions to nephrology.