Maia Parsadanian

发表

Human Immunodeficiency Virus Type 1 Alters Brain-Derived Neurotrophic Factor Processing in Neurons

L. Zecca, M. Parsadanian, A. Bachis, 2012, The Journal of Neuroscience.

Plaque‐associated disruption of CSF and plasma amyloid‐β (Aβ) equilibrium in a mouse model of Alzheimer's disease

D. Holtzman, K. Bales, R. Demattos, 2002, Journal of neurochemistry.

Overexpression of ABCA1 reduces amyloid deposition in the PDAPP mouse model of Alzheimer disease.

D. Holtzman, S. Paul, C. Wellington, 2008, The Journal of clinical investigation.

Deletion of Abca1 Increases Aβ Deposition in the PDAPP Transgenic Mouse Model of Alzheimer Disease*

D. Holtzman, S. Paul, R. Hartman, 2005, Journal of Biological Chemistry.

Apolipoprotein E isoform-dependent amyloid deposition and neuritic degeneration in a mouse model of Alzheimer's disease.

A. Fagan, D. Holtzman, D. Holtzman, 2000, Proceedings of the National Academy of Sciences of the United States of America.

Human Apolipoprotein E4 Alters the Amyloid-β 40:42 Ratio and Promotes the Formation of Cerebral Amyloid Angiopathy in an Amyloid Precursor Protein Transgenic Model

D. Holtzman, S. Paul, K. Bales, 2005, The Journal of Neuroscience.

ApoE and Clusterin Cooperatively Suppress Aβ Levels and Deposition Evidence that ApoE Regulates Extracellular Aβ Metabolism In Vivo

Bruce J Aronow, D. Holtzman, S. Paul, 2004, Neuron.

In Vivo Assessment of Brain Interstitial Fluid with Microdialysis Reveals Plaque-Associated Changes in Amyloid-β Metabolism and Half-Life

D. Holtzman, S. Paul, J. Cirrito, 2003, The Journal of Neuroscience.

Apolipoprotein E Markedly Facilitates Age-Dependent Cerebral Amyloid Angiopathy and Spontaneous Hemorrhage in Amyloid Precursor Protein Transgenic Mice

D. Holtzman, S. Paul, K. Bales, 2003, The Journal of Neuroscience.

Clusterin promotes amyloid plaque formation and is critical for neuritic toxicity in a mouse model of Alzheimer's disease

Bruce J Aronow, David M Holtzman, D. Holtzman, 2002, Proceedings of the National Academy of Sciences of the United States of America.

Neuronal enhancers are hotspots for DNA single-strand break repair

Michael E. Ward, A. Nussenzweig, P. McHugh, 2020, Nature.

ABCA1 Is Required for Normal Central Nervous System ApoE Levels and for Lipidation of Astrocyte-secreted apoE*

Xianlin Han, D. Holtzman, T. Kowalewski, 2004, Journal of Biological Chemistry.

BDNF Blocks Caspase-3 Activation in Neonatal Hypoxia–Ischemia

A. Shah, D. Holtzman, S. Back, 2000, Neurobiology of Disease.

Rapid Microglial Response Around Amyloid Pathology after Systemic Anti-Aβ Antibody Administration in PDAPP Mice

D. Holtzman, B. Hyman, M. Meyer-Luehmann, 2008, The Journal of Neuroscience.

BAX Contributes to Apoptotic-Like Death Following Neonatal Hypoxia-Ischemia: Evidence for Distinct Apoptosis Pathways

D. Holtzman, C. Knudson, Junjeong Choi, 2001, Molecular medicine.

Plaque-associated disruption of CSF and plasma amyloid-beta (Abeta) equilibrium in a mouse model of Alzheimer's disease.

D. Holtzman, S. Paul, K. Bales, 2002, Journal of neurochemistry.

Expression of human apolipoprotein E reduces amyloid-beta deposition in a mouse model of Alzheimer's disease.

A. Fagan, D. Holtzman, D. Holtzman, 1999, The Journal of clinical investigation.

Overexpression of Low-Density Lipoprotein Receptor in the Brain Markedly Inhibits Amyloid Deposition and Increases Extracellular Aβ Clearance

D. Holtzman, S. Paul, Hong Jiang, 2009, Neuron.

Electromagnetic controlled cortical impact device for precise, graded experimental traumatic brain injury.

Philip V Bayly, Chad C. Kessens, David L Brody, 2007, Journal of neurotrauma.

Measurement of apolipoprotein E and amyloid β clearance rates in the mouse brain using bolus stable isotope labeling

D. Holtzman, R. Bateman, Hong Jiang, 2012, Molecular Neurodegeneration.

P-glycoprotein deficiency at the blood-brain barrier increases amyloid-beta deposition in an Alzheimer disease mouse model.

A. Fagan, D. Holtzman, S. Paul, 2005, The Journal of clinical investigation.

Human and Murine ApoE Markedly Alters Aβ Metabolism before and after Plaque Formation in a Mouse Model of Alzheimer's Disease

A. Fagan, D. Holtzman, S. Paul, 2002, Neurobiology of Disease.

ApoAI deficiency results in marked reductions in plasma cholesterol but no alterations in amyloid-beta pathology in a mouse model of Alzheimer's disease-like cerebral amyloidosis.

A. Fagan, D. Holtzman, S. Paul, 2004, The American journal of pathology.

Experimental traumatic brain injury induces rapid aggregation and oligomerization of amyloid-beta in an Alzheimer's disease mouse model.

Maia Parsadanian, M. Parsadanian, Mark P Burns, 2014, Journal of neurotrauma.

Correction: In Vivo Human Apolipoprotein E Isoform Fractional Turnover Rates in the CNS

D. Holtzman, R. Bateman, C. Xiong, 2012, PLoS ONE.

In Vivo Human Apolipoprotein E Isoform Fractional Turnover Rates in the CNS

D. Holtzman, R. Bateman, C. Xiong, 2012, PloS one.

Dendritic Spine Loss and Chronic White Matter Inflammation in a Mouse Model of Highly Repetitive Head Trauma.

S. Villapol, Charisse N. Winston, M. Parsadanian, 2016, The American journal of pathology.

The Low Density Lipoprotein Receptor Regulates the Level of Central Nervous System Human and Murine Apolipoprotein E but Does Not Modify Amyloid Plaque Pathology in PDAPP Mice*

D. Holtzman, S. Paul, G. Bu, 2005, Journal of Biological Chemistry.

Apolipoprotein E4 impairs spontaneous blood brain barrier repair following traumatic brain injury

S. Villapol, Olga C. Rodriguez, M. Burns, 2018, Molecular Neurodegeneration.

P4-358 In vitro and in vivo characterization of beta-amyloid antibodies binding to cerebral amyloid angiopathy (CAA) and the selective exacerbation of CAA-associated microhemorrhage

D. Holtzman, S. Paul, K. Bales, 2004, Neurobiology of Aging.

Neurotrophins modulate the expression of chemokine receptors in the brain

L. Tessarollo, T. Mhyre, I. Mocchetti, 2011, Journal of Neurovirology.

Reduced cortical excitatory synapse number in APOE4 mice is associated with increased calcineurin activity

S. Villapol, Charisse N. Winston, M. Parsadanian, 2017, Neuroreport.

The effect of injury severity on behavior: a phenotypic study of cognitive and emotional deficits after mild, moderate, and severe controlled cortical impact injury in mice.

P. Forcelli, M. Parsadanian, David N Zapple, 2012, Journal of neurotrauma.