Zehra Kevser Timur

Murine Sialidase Neu3 facilitates GM2 degradation and bypass in mouse model of Tay-Sachs disease

E. Oztaş, C. Hopf, K. Yamaguchi, 2018, Experimental Neurology.

Sialidase neu4 deficiency is associated with neuroinflammation in mice

V. Seyrantepe, S. Demir, Z. K. Timur, 2021, Glycoconjugate Journal.

Lysosomal Cathepsin A Plays a Significant Role in the Processing of Endogenous Bioactive Peptides

V. Seyrantepe, Z. K. Timur, S. Akyildiz Demir, 2016, Front. Mol. Biosci..

GM2 ganglioside accumulation causes neuroinflammation and behavioral alterations in a mouse model of early onset Tay-Sachs disease

V. Seyrantepe, S. Demir, Z. K. Timur, 2020, Journal of neuroinflammation.

Neuraminidase-1 contributes significantly to the degradation of neuronal B-series gangliosides but not to the bypass of the catabolic block in Tay–Sachs mouse models

R. Sandhoff, C. Marsching, V. Seyrantepe, 2015, Molecular genetics and metabolism reports.