L. D. Kerr

发表

Vascular endothelial growth factor affects dendritic cell maturation through the inhibition of nuclear factor-kappa B activation in hemopoietic progenitor cells.

D. Carbone, T. Oyama, S. Nadaf, 1998, Journal of immunology.

Rel/NF-KB Represses bcl-2 Transcription in pro-B Lym phocytes

M. Byrom, Chih‐Li Chen, L. Kerr, 2016 .

Multiorgan nuclear factor kappa B activation in a transgenic mouse model of systemic inflammation.

J. Christman, L. Lancaster, T. Blackwell, 2000, American journal of respiratory and critical care medicine.

Nuclear factor-kappaB (NF-kappaB) regulates proliferation and branching in mouse mammary epithelium.

F. Kittrell, L. Matrisian, D. Medina, 2001, Molecular biology of the cell.

Nuclear Factor-κB (NF-κB) Regulates Proliferation and Branching in Mouse Mammary Epithelium

F. Kittrell, L. Matrisian, D. Medina, 2001 .

c-rel activates but v-rel suppresses transcription from kappa B sites.

T. Hunter, I. Verma, J. Inoue, 1991, Proceedings of the National Academy of Sciences of the United States of America.

Activation of IL-8 gene expression by Helicobacter pylori is regulated by transcription factor nuclear factor-kappa B in gastric epithelial cells.

M. Blaser, M. Tummuru, L. Kerr, 1998, Journal of immunology.

Association between proto-oncoprotein Rel and TATA-binding protein mediates transcriptional activation by NF-κB

Qiang Zhou, A. Berk, T. Boyer, 1993, Nature.

IκBγ, a 70 kd protein identical to the C-terminal half of p110 NF-κB: A new member of the IκB family

I. Verma, J. Inoue, A. Kakizuka, 1992, Cell.

The rel-associated pp40 protein prevents DNA binding of Rel and NF-kappa B: relationship with I kappa B beta and regulation by phosphorylation.

I. Verma, H. Bose,, J. Inoue, 1991, Genes & development.

Signal transduction: the nuclear target.

I. Verma, J. Inoue, Inder M. Verma, 1992, Current opinion in cell biology.

Lymphocytes Lacking IκB-α Develop Normally, But Have Selective Defects in Proliferation and Function1

F. Yull, Nagendra Singh, L. Van Kaer, 2000, The Journal of Immunology.

The role of C-Fos in growth factor regulation of stromelysin/transin gene expression.

L. Matrisian, B. Magun, L. Kerr, 1992, Matrix (Stuttgart, Germany). Supplement.

Growth factors regulate transin gene expression by c-fos-dependent and c-fos-independent pathways.

L. Matrisian, J. Holt, L. Kerr, 1988, Science.

Expression of Glutathione and γ-Glutamylcysteine Synthetase mRNA Is Jun Dependent

D. Spitz, M. Freeman, K. Sekhar, 1997 .

TGF-β1 inhibition of transin/stromelysin gene expression is mediated through a fos binding sequence

L. Matrisian, L. Kerr, Donna B. Miller, 1990, Cell.

Protein kinase C isotypes required for phorbol‐ester induction of stromelysin‐1 in rat fibroblasts

L. Matrisian, L. Kerr, M. Gaire, 1996, Molecular carcinogenesis.

Negative regulation of gene expression by TGF‐β

L. Matrisian, L. Wood, L. Kerr, 1992 .

The proto-oncogene bcl-3 encodes an I kappa B protein.

G. Nabel, I. Verma, C. Duckett, 1992, Genes & development.

Dynamic expression and activity of NF-κB during post-natal mammary gland morphogenesis

R. Muraoka, D. Brantley, F. Yull, 2000, Mechanisms of Development.

Use of genetically altered mice to investigate the role of nuclear factor-kappa B activation and cytokine gene expression in sepsis-induced ARDS.

J. Christman, L. Lancaster, T. Blackwell, 1999, Chest.

The proto-oncogene BCL-3 encodes an IκB protein

G. Nabel, I. Verma, C. Duckett, 1992 .

Transforming growth factor beta 1 and cAMP inhibit transcription of epidermal growth factor- and oncogene-induced transin RNA.

L. Matrisian, N. Olashaw, L. Kerr, 1988, The Journal of biological chemistry.

Epidermal growth factor stimulation of stromelysin mRNA in rat fibroblasts requires induction of proto-oncogenes c-fos and c-jun and activation of protein kinase C

L. Matrisian, S. Mcdonnell, L. Kerr, 1990, Molecular and cellular biology.

Negative regulation of gene expression by TGF-beta.

L. Wood, G. Ganser, L. Matrisian, 1992, Molecular reproduction and development.

RAG2-/-, I kappa B-alpha-/- chimeras display a psoriasiform skin disease.

F. Yull, L. Nanney, C. L. Chen, 2000, The Journal of investigative dermatology.

The bZIP domains of Fos and Jun mediate a physical association with the TATA box-binding protein.

I. Verma, L. Ransone, L. Kerr, 1993, Gene expression.

Arrested development: understanding v-abl.

L. Kerr, L. D. Kerr, 1994, BioEssays : news and reviews in molecular, cellular and developmental biology.

Inhibition of NF-κB activity results in disruption of the apical ectodermal ridge and aberrant limb morphogenesis

D. Brantley, F. Yull, L. Niswander, 1998, Nature.

Reovirus-Induced Apoptosis Requires Activation of Transcription Factor NF-κB

K. Tyler, T. Dermody, D. Ballard, 2000, Journal of Virology.

AD Award Number : DAMD 1797-1-7017 TITLE : The Role of NF-kB in Normal and Transformed Mammary Epithelium

D. Brantley, L. Kerr, L. D. Kerr, 2022 .

Expression of glutathione and gamma-glutamylcysteine synthetase mRNA is Jun dependent.

D. Spitz, M. Freeman, K. Sekhar, 1997, Biochemical and biophysical research communications.

I kappa B gamma, a 70 kd protein identical to the C-terminal half of p110 NF-kappa B: a new member of the I kappa B family.

I. Verma, J. Inoue, A. Kakizuka, 1992, Cell.