Diana Wagner

Wallerian degeneration of injured axons and synapses is delayed by a Ube4b/Nmnat chimeric gene

V. Perry, C. Andressen, R. Ribchester, 2001, Nature Neuroscience.

The progressive nature of Wallerian degeneration in wild-type and slow Wallerian degeneration (WldS) nerves

R. Ribchester, B. Beirowski, R. Adalbert, 2005, BMC Neuroscience.

Quantitative and qualitative analysis of Wallerian degeneration using restricted axonal labelling in YFP-H mice

Richard R. Ribchester, Michael P. Coleman, R. Ribchester, 2004, Journal of Neuroscience Methods.

Proteasome inhibition arrests neurite outgrowth and causes “dying‐back” degeneration in primary culture

D. Wagner, Till G. A. Mack, H. Laser, 2003, Journal of neuroscience research.

The slow Wallerian degeneration gene, WldS, inhibits axonal spheroid pathology in gracile axonal dystrophy mice.

Hitoshi Osaka, R. Ribchester, T. Gillingwater, 2004, Brain : a journal of neurology.

The slow Wallerian degeneration protein, WldS, binds directly to VCP/p97 and partially redistributes it within the nucleus.

R. Ribchester, J. Haley, G. Morreale, 2005, Molecular biology of the cell.

A rat model of slow Wallerian degeneration (WldS) with improved preservation of neuromuscular synapses

R. Ribchester, T. Gillingwater, J. Haley, 2005, The European journal of neuroscience.

A Ufd2/D4Cole1e chimeric protein and overexpression of Rbp7 in the slow Wallerian degeneration (WldS) mouse.

V. Perry, D. Wagner, M. Coleman, 2000, Proceedings of the National Academy of Sciences of the United States of America.

Reducing expression of NAD+ synthesizing enzyme NMNAT1 does not affect the rate of Wallerian degeneration

G. Magni, C. Bendotti, M. Coleman, 2011, The FEBS journal.