Eckard Hamelmann

发表

Passive transfer of immediate hypersensitivity and airway hyperresponsiveness by allergen-specific immunoglobulin (Ig) E and IgG1 in mice.

E. Gelfand, E. Hamelmann, G. Larsen, 1996, The Journal of clinical investigation.

Dynamic evolution of serum immunoglobulin E to airborne allergens throughout childhood: results from the Multi‐Centre Allergy Study birth cohort

B. Niggemann, T. Keil, S. Lau, 2009, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.

Local treatment with IL-12 is an effective inhibitor of airway hyperresponsiveness and lung eosinophilia after airway challenge in sensitized mice.

E. Gelfand, E. Hamelmann, G. Cieslewicz, 1998, The Journal of allergy and clinical immunology.

Development of eosinophilic airway inflammation and airway hyperresponsiveness requires interleukin-5 but not immunoglobulin E or B lymphocytes.

E. Gelfand, E. Hamelmann, J. Schwarze, 1999, American journal of respiratory cell and molecular biology.

IL-5 and eosinophils are essential for the development of airway hyperresponsiveness following acute respiratory syncytial virus infection.

E. Gelfand, E. Hamelmann, G. Cieslewicz, 1999, Journal of immunology.

Allergic airway sensitization induces T cell activation but not airway hyperresponsiveness in B cell-deficient mice.

P. Marrack, E. Gelfand, J. Kappler, 1997, Proceedings of the National Academy of Sciences of the United States of America.

Nebulized IFN-gamma inhibits the development of secondary allergic responses in mice.

E. Gelfand, E. Hamelmann, G. Lack, 1996, Journal of immunology.

Therapeutic potential of larval excretory/secretory proteins of the pig whipworm Trichuris suis in allergic disease

S. Hartmann, S. Rausch, A. Kühl, 2014, Allergy.

Development of eosinophilic airway inflammation and airway hyperresponsiveness requires interleukin-5 but not immunoglobulin E or B lymphocytes.

E. Gelfand, J. Schwarze, C. Irvin, 1999, American journal of respiratory cell and molecular biology.

Antigen-specific immunoglobulin-A prevents increased airway responsiveness and lung eosinophilia after airway challenge in sensitized mice.

E. Gelfand, E. Hamelmann, G. Cieslewicz, 1998, American journal of respiratory and critical care medicine.

Anti-interleukin 5 but not anti-IgE prevents airway inflammation and airway hyperresponsiveness.

T. Ishizuka, E. Gelfand, E. Hamelmann, 1999, American journal of respiratory and critical care medicine.

CD8 T cells are essential in the development of respiratory syncytial virus-induced lung eosinophilia and airway hyperresponsiveness.

E. Gelfand, E. Hamelmann, G. Cieslewicz, 1999, Journal of immunology.

Infection Following Acute Respiratory Syncytial Virus Development of Airway Hyperresponsiveness IL-5 and Eosinophils Are Essential for the and

E. Gelfand, E. Hamelmann, G. Cieslewicz, 1999 .

Allergen-specific IgE and IL-5 are essential for the development of airway hyperresponsiveness.

E. Gelfand, E. Hamelmann, J. Schwarze, 1997, American journal of respiratory cell and molecular biology.

CD23 deficient mice develop allergic airway hyperresponsiveness following sensitization with ovalbumin.

E. Hamelmann, A. Joetham, H. Kikutani, 1997, American journal of respiratory and critical care medicine.

Anti-CD86 (B7.2) treatment abolishes allergic airway hyperresponsiveness in mice.

E. Hamelmann, G. Cieslewicz, A. Joetham, 1999, American journal of respiratory and critical care medicine.

BCG Priming of Dendritic Cells Enhances T Regulatory and Th1 Function and Suppresses Allergen-Induced Th2 Function in vitro and in vivo

U. Wahn, E. Hamelmann, D. Quarcoo, 2009, International Archives of Allergy and Immunology.

Primary prevention of allergy: avoiding risk or providing protection?

B. Niggemann, S. Lau, U. Wahn, 2007, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.

Master switches of T-cell activation and differentiation

E. Hamelmann, T. Kallinich, K. Beier, 2007, European Respiratory Journal.