F. Hughes

发表

Inflammasomes in the urinary tract: a disease-based review.

F. Hughes, J. Purves, J. Purves, 2016, American journal of physiology. Renal physiology.

Specialized pro-resolution mediators in the bladder: Receptor expression and recovery of bladder function from cystitis

Huixia Jin, J. Purves, Francis M Hughes, 2022, Experimental biology and medicine.

A Highly Polarized Th2 Bladder Response to Infection Promotes Epithelial Repair at the Expense of Preventing New Infections

C. Phoenix, Y. Miao, Jianxuan Wu, 2020, Nature Immunology.

NLRP3 Promotes Diabetic Bladder Dysfunction and Changes in Symptom-Specific Bladder Innervation

Nathan Hirshman, F. Hughes, Huixia Jin, 2018, Diabetes.

Why Are Some People with Lower Urinary Tract Symptoms (LUTS) Depressed? New Evidence That Peripheral Inflammation in the Bladder Causes Central Inflammation and Mood Disorders

J. Purves, Francis M Hughes, M. R. Odom, 2023, International journal of molecular sciences.

Inflammation triggered by the NLRP3 inflammasome is a critical driver of diabetic bladder dysfunction

J. Purves, Francis M Hughes, M. R. Odom, 2022, Frontiers in Physiology.

The potential repertoire of the innate immune system in the bladder: expression of pattern recognition receptors in the rat bladder and a rat urothelial cell line (MYP3 cells)

David P Turner, D. Turner, J. Purves, 2015, International Urology and Nephrology.

Inflammasomes are important mediators of cyclophosphamide-induced bladder inflammation.

P. Nietert, F. Hughes, J. Purves, 2014, American journal of physiology. Renal physiology.

Recurrent infections drive persistent bladder dysfunction and pain via sensory nerve sprouting and mast cell activity

J. Purves, Chunjing Bao, Y. Huh, 2024, Science Immunology.

NLRP3/IL‐1β mediates denervation during bladder outlet obstruction in rats

F. Hughes, Huixia Jin, J. Purves, 2018, Neurourology and urodynamics.

The NLRP3 Inflammasome Mediates Inflammation Produced by Bladder Outlet Obstruction.

W. Foo, F. Hughes, J. Purves, 2016, The Journal of urology.

Diabetic bladder dysfunction progresses from an overactive to an underactive phenotype in a type-1 diabetic mouse model (Akita female mouse) and is dependent on NLRP3.

Huixia Jin, J. Purves, Francis M Hughes, 2022, Life sciences.

Strict glucose control and elimination of NLRP3-induced inflammation prevents diabetic bladder dysfunction in the female Akita mouse model.

Huixia Jin, Shelby N Harper, J. Todd Purves, 2024, Neurourology and urodynamics.

Specialized pro-resolution mediators in the bladder: effects of resolvin E1 on diabetic bladder dysfunction in the type 1 diabetic male Akita mouse model

Huixia Jin, J. Purves, Anissa Cervantes, 2024, BMC urology.

Female Type 1 Diabetic Akita Mice Demonstrate Increased Bladder Contractility via FP Receptor Activation due to NLRP3-Mediated Inflammation.

Huixia Jin, J. Purves, F. Hughes, 2024, Frontiers in bioscience.

Male Akita diabetic mice develop underactive bladder independent of NLRP3 that can be prevented with blood glucose control

Huixia Jin, J. Purves, F. Hughes, 2024, Continence.