Amy Cook

发表

Enhanced targeting of CML stem and progenitor cells by inhibition of porcupine acyltransferase in combination with TKI.

Youzhen Wang, M. Mclaughlin, F. Mikhail, 2017, Blood.

JAK2/STAT5 inhibition by nilotinib with ruxolitinib contributes to the elimination of CML CD34+ cells in vitro and in vivo.

Susan Rhodes, Anthony D. Whetton, T. Holyoake, 2014, Blood.

JAK 2 / STAT 5 inhibition by nilotinib with ruxolitinib contributes to the elimination of chronic phase CML CD 34 + cells in vitro and in vivo

T. Holyoake, L. Hopcroft, S. Rhodes, 2014 .

Role of altered growth factor receptor-mediated JAK2 signaling in growth and maintenance of human acute myeloid leukemia stem cells.

R. Jove, D. Perrotti, S. Forman, 2014, Blood.

The Death Domain Kinase RIP Is Essential for TRAIL (Apo2L)-Induced Activation of IκB Kinase and c-Jun N-Terminal Kinase

M. Kelliher, S. Lipkowitz, M. Keane, 2000, Molecular and Cellular Biology.

Role of c-Jun N-terminal kinase in apoptosis.

T. H. Wang, Z. Liu, A. Cook, 2001, Methods in cell biology.

MYELOID NEOPLASIA JAK2/STAT5 inhibition by nilotinib with ruxolitinib contributes to the elimination of CML CD34 + cells in vitro and in vivo

T. Holyoake, A. Whetton, L. Hopcroft, 2014 .

growth and maintenance of human acute myeloid leukemia stem cells Role of altered growth factor receptor-mediated JAK2 signaling in

R. Jove, S. Forman, R. Bhatia, 2014 .

Enhanced targeting of CML stem and progenitor cells by inhibition of porcupine acyltransferase in combination with TKI.

Youzhen Wang, M. Mclaughlin, F. Mikhail, 2017, Blood.