Rafael Ikemori

发表

Epigenetic Reprogramming of Tumor-Associated Fibroblasts in Lung Cancer: Therapeutic Opportunities

J. Alcaraz, N. Reguart, M. Vizoso, 2021, Cancers.

Nintedanib selectively inhibits the activation and tumour-promoting effects of fibroblasts from lung adenocarcinoma patients

J. Alcaraz, N. Reguart, F. Hilberg, 2017, British Journal of Cancer.

Aberrant TIMP-1 overexpression in tumor-associated fibroblasts drives tumor progression through CD63 in lung adenocarcinoma.

J. Alcaraz, D. Radisky, H. Mori, 2022, Matrix biology : journal of the International Society for Matrix Biology.

Epigenetic SMAD3 repression in tumor-associated fibroblasts impairs fibrosis and response to the antifibrotic drug nintedanib in lung squamous cell carcinoma.

J. Alcaraz, M. Esteller, V. Peinado, 2019, Cancer research.

MMP1 drives tumor progression in large cell carcinoma of the lung through fibroblast senescence.

J. Alcaraz, D. Radisky, P. Gascón, 2021, Cancer letters.

Characterization of cells recovered from the xenotransplanted NG97 human-derived glioma cell line subcultured in a long-term in vitro

W. Savino, L. Verinaud, A. Schenka, 2008, BMC Cancer.

Galectin-3 Up-Regulation in Hypoxic and Nutrient Deprived Microenvironments Promotes Cell Survival

S. Nonogaki, K. Umezawa, E. Osinaga, 2014, PloS one.

3D collagen migration patterns reveal a SMAD3-dependent and TGF-β1-independent mechanism of recruitment for tumour-associated fibroblasts in lung adenocarcinoma

J. Alcaraz, J. Ramírez, N. Reguart, 2022, British Journal of Cancer.

Nintedanib selectively inhibits the activation and tumour-promoting effects of fibroblasts from lung adenocarcinoma patients

J. Alcaraz, N. Reguart, F. Hilberg, 2017, British Journal of Cancer.

Aberrant TIMP‐1 production in tumor‐associated fibroblasts drives the selective benefits of nintedanib in lung adenocarcinoma

J. Alcaraz, D. Radisky, F. Hilberg, 2024, Cancer science.