Salvatore Nesci

发表

Effects of Hydrogen Sulfide Donor NaHS on Porcine Vascular Wall-Mesenchymal Stem Cells

M. Forni, A. Zannoni, Cristina Algieri, 2020, International journal of molecular sciences.

GPR35, ally of the anti-ischemic ATPIF1-ATP synthase interaction.

S. Nesci, Salvatore Nesci, 2022, Trends in pharmacological sciences.

Emerging Roles for the Mitochondrial ATP Synthase Supercomplexes.

S. Nesci, A. Pagliarani, Salvatore Nesci, 2019, Trends in biochemical sciences.

SARS‐CoV‐2 first contact: Spike–ACE2 interactions in COVID‐19

Salvatore Nesci, 2021, Chemical biology & drug design.

A Therapeutic Role for the F1FO-ATP Synthase

Cristina Algieri, F. Trombetti, S. Nesci, 2019, SLAS discovery : advancing life sciences R & D.

Opposite Rotation Directions in the Synthesis and Hydrolysis of ATP by the ATP Synthase: Hints from a Subunit Asymmetry

F. Trombetti, S. Nesci, A. Pagliarani, 2015, The Journal of Membrane Biology.

Thiol oxidation of mitochondrial F0-c subunits: a way to switch off antimicrobial drug targets of the mitochondrial ATP synthase.

F. Trombetti, S. Nesci, A. Pagliarani, 2014, Medical hypotheses.

Thiol oxidation is crucial in the desensitization of the mitochondrial F1FO-ATPase to oligomycin and other macrolide antibiotics.

F. Trombetti, S. Nesci, A. Pagliarani, 2014, Biochimica et biophysica acta.

Modifiers of the oligomycin sensitivity of the mitochondrial F1F0-ATPase.

S. Nesci, A. Pagliarani, V. Ventrella, 2013, Mitochondrion.

F. Trombetti, S. Nesci, A. Pagliarani, 2016 .

The a subunit asymmetry dictates the two opposite rotation directions in the synthesis and hydrolysis of ATP by the mitochondrial ATP synthase.

F. Trombetti, S. Nesci, A. Pagliarani, 2015, Medical hypotheses.

Post-translational modifications of the mitochondrial F1FO-ATPase.

F. Trombetti, S. Nesci, A. Pagliarani, 2017, Biochimica et biophysica acta. General subjects.

Kinetic properties of the mitochondrial F1FO-ATPase activity elicited by Ca2+ in replacement of Mg2.

F. Trombetti, S. Nesci, A. Pagliarani, 2017, Biochimie.

Preferential nitrite inhibition of the mitochondrial F1FO-ATPase activities when activated by Ca(2+) in replacement of the natural cofactor Mg(2+).

F. Trombetti, S. Nesci, A. Pagliarani, 2016, Biochimica et biophysica acta.

Molecular and Supramolecular Structure of the Mitochondrial Oxidative Phosphorylation System: Implications for Pathology

G. Lenaz, Cristina Algieri, F. Trombetti, 2021, Life.

From the Structural and (Dys)Function of ATP Synthase to Deficiency in Age-Related Diseases

C. Garone, A. Pietra, S. Nesci, 2022, Life.

Cellular Metabolism and Bioenergetic Function in Human Fibroblasts and Preadipocytes of Type 2 Familial Partial Lipodystrophy

M. Forni, A. Zannoni, Cristina Algieri, 2022, International journal of molecular sciences.

Characterization of metabolic profiles and lipopolysaccharide effects on porcine vascular wall mesenchymal stem cells

M. Forni, A. Zannoni, C. Bernardini, 2019, Journal of cellular physiology.

Proton leak through the UCPs and ANT carriers and beyond: A breath for the electron transport chain.

Salvatore Nesci, 2023, Biochimie.

Inflammation, Mitochondria and Natural Compounds Together in the Circle of Trust

F. Oppedisano, S. Nesci, A. Spagnoletta, 2023, International journal of molecular sciences.

Nicotinamide Nucleotide Transhydrogenase as a Sensor of Mitochondrial Biology.

F. Trombetti, S. Nesci, A. Pagliarani, 2019, Trends in cell biology.

The mitochondrial permeability transition pore in cell death: A promising drug binding bioarchitecture

S. Nesci, Salvatore Nesci, 2020, Medicinal research reviews.

Cellular metabolism therapy

S. Nesci, Salvatore Nesci, 2022, Journal of translational medicine.

Dietary enhancement of selected fatty acid biosynthesis in the digestive gland of Mytilus galloprovincialis Lmk.

F. Trombetti, S. Nesci, A. Pagliarani, 2013, Journal of agricultural and food chemistry.

From the Ca2+-activated F1FO-ATPase to the mitochondrial permeability transition pore: an overview.

F. Trombetti, S. Nesci, A. Pagliarani, 2018, Biochimie.

Two separate pathways underlie NADH and succinate oxidation in swine heart mitochondria: Kinetic evidence on the mobile electron carriers.

G. Lenaz, M. Fabbri, Cristina Algieri, 2023, Biochimica et biophysica acta. Bioenergetics.

Novel Regioselective Synthesis of 1,3,4,5-Tetrasubstituted Pyrazoles and Biochemical Valuation on F1FO-ATPase and Mitochondrial Permeability Transition Pore Formation

P. Costanzo, L. Maiuolo, Vincenzo Algieri, 2023, Pharmaceutics.

Biological characteristics and metabolic profile of canine mesenchymal stem cells isolated from adipose tissue and umbilical cord matrix

Cristina Algieri, S. Nesci, A. Pagliarani, 2021, PloS one.

Mercury and protein thiols: Stimulation of mitochondrial F1FO-ATPase and inhibition of respiration.

F. Trombetti, S. Nesci, A. Pagliarani, 2016, Chemico-biological interactions.

Inflammatory signaling in NASH driven by hepatocyte mitochondrial dysfunctions

V. De Giorgi, Salvatore Nesci, Francesca Oppedisano, 2023, Journal of Translational Medicine.

The mitochondrial F1FO-ATPase exploits the dithiol redox state to modulate the permeability transition pore.

Cristina Algieri, F. Trombetti, S. Nesci, 2021, Archives of biochemistry and biophysics.

Ca2+ as cofactor of the mitochondrial H+‐translocating F1FO‐ATP(hydrol)ase

A. Pagliarani, Salvatore Nesci, 2020, Proteins.

Mineralocorticoid Receptor Antagonism Prevents Type 2 Familial Partial Lipodystrophy Brown Adipocyte Dysfunction

E. Gabusi, G. Lisignoli, M. Caprio, 2023, Cells.

Salvatore Nesci, Giovanni Romeo, 2024, Medicinal research reviews.

Crucial aminoacids in the FO sector of the F1FO-ATP synthase address H+ across the inner mitochondrial membrane: molecular implications in mitochondrial dysfunctions

Cristina Algieri, F. Trombetti, S. Nesci, 2019, Amino Acids.

Incoming news on the F-type ATPase structure and functions in mammalian mitochondria.

S. Nesci, A. Pagliarani, Salvatore Nesci, 2021, BBA advances.

Relationship between serum concentration, functional parameters and cell bioenergetics in IPEC-J2 cell line

M. Forni, A. Zannoni, Cristina Algieri, 2021, Histochemistry and Cell Biology.

Mussel and mammalian ATP synthase share the same bioenergetic cost of ATP

F. Trombetti, S. Nesci, A. Pagliarani, 2013, Journal of Bioenergetics and Biomembranes.

Enjoy your journey: the bergamot polyphenols from the tree to the cell metabolism

V. Mollace, E. Palma, F. Oppedisano, 2021, Journal of translational medicine.

What happens when the mitochondrial H+-translocating F1FO-ATP(hydrol)ase becomes a molecular target of calcium? The pore opens.

S. Nesci, Salvatore Nesci, 2022, Biochimie.

Modifiers of the oligomycin sensitivity of the mitochondrial F1F0-ATPase.

A. Pagliarani, V. Ventrella, Salvatore Nesci, 2013, Mitochondrion.

Mitochondrial Ca2+‐activated F1FO‐ATPase hydrolyzes ATP and promotes the permeability transition pore

M. Forni, M. Fabbri, Cristina Algieri, 2019, Annals of the New York Academy of Sciences.

Phenylglyoxal inhibition of the mitochondrial F1FO-ATPase activated by Mg2+ or by Ca2+ provides clues on the mitochondrial permeability transition pore.

Cristina Algieri, F. Trombetti, S. Nesci, 2020, Archives of biochemistry and biophysics.

inhibition of the mitochondrial F 1 F O -ATPase activated by Mg 2+ or by Ca 2+ provides clues on the mitochondrial permeability transition pore

Cristina Algieri, F. Trombetti, A. Pagliarani, 2022 .

New insight in a new entity: the mitochondrial permeability transition pore arises from the Ca2+-activated F1FO-ATPases.

S. Nesci, Salvatore Nesci, 2017, Science bulletin.

The mitochondrial F1FO-ATPase desensitization to oligomycin by tributyltin is due to thiol oxidation.

F. Trombetti, S. Nesci, A. Pagliarani, 2014, Biochimie.

Tributyltin inhibits the oligomycin-sensitive Mg-ATPase activity in Mytilus galloprovincialis digestive gland mitochondria.

F. Trombetti, S. Nesci, A. Pagliarani, 2011, Comparative biochemistry and physiology. Toxicology & pharmacology : CBP.

Tributyltin (TBT) and dibutyltin (DBT) differently inhibit the mitochondrial Mg-ATPase activity in mussel digestive gland.

F. Trombetti, A. Pagliarani, M. Pirini, 2011, Toxicology in vitro : an international journal published in association with BIBRA.

Glucose and glutamine in the mitochondrial oxidative metabolism of stem cells.

Salvatore Nesci, 2017, Mitochondrion.

Mitochondrial sirtuin 3 and role of natural compounds: the effect of post-translational modifications on cellular metabolism.

A. Spagnoletta, Salvatore Nesci, Francesca Oppedisano, 2024, Critical reviews in biochemistry and molecular biology.

UCP2, a Member of the Mitochondrial Uncoupling Proteins: An Overview from Physiological to Pathological Roles

Salvatore Nesci, Speranza Rubattu, S. Rubattu, 2024, Biomedicines.

Vitamin K Vitamers Differently Affect Energy Metabolism in IPEC-J2 Cells

M. Forni, Cristina Algieri, C. Bernardini, 2021, Frontiers in Molecular Biosciences.

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F. Trombetti, A. Pagliarani, Salvatore Nesci, 2020 .

Season and Cooking May Alter Fatty Acids Profile of Polar Lipids from Blue-Back Fish.

F. Farabegoli, A. Badiani, S. Nesci, 2019, Lipids.

1,5-disubstituted-1,2,3-triazoles counteract mitochondrial dysfunction acting on F1FO-ATPase in models of cardiovascular diseases.

M. Forni, S. Sciarretta, P. Costanzo, 2022, Pharmacological research.

1,5‐Disubstituted‐1,2,3‐triazoles as inhibitors of the mitochondrial Ca2+‐activated F1FO‐ATP(hydrol)ase and the permeability transition pore

L. Maiuolo, Vincenzo Algieri, Cristina Algieri, 2020, Annals of the New York Academy of Sciences.

Molecular mechanisms of naringenin modulation of mitochondrial permeability transition acting on F1FO-ATPase and counteracting saline load-induced injury in SHRSP cerebral endothelial cells.

S. Marchi, R. Bigi, L. Maiuolo, 2024, European journal of cell biology.

Toxicity of organotin compounds: shared and unshared biochemical targets and mechanisms in animal cells.

S. Nesci, A. Pagliarani, V. Ventrella, 2013, Toxicology in vitro : an international journal published in association with BIBRA.

The inhibition of the mitochondrial F1FO-ATPase activity when activated by Ca2+ opens new regulatory roles for NAD+

F. Trombetti, S. Nesci, A. Pagliarani, 2018, Biological chemistry.

The Impairment of Cell Metabolism by Cardiovascular Toxicity of Doxorubicin Is Reversed by Bergamot Polyphenolic Fraction Treatment in Endothelial Cells

M. Forni, V. Mollace, E. Palma, 2022, International journal of molecular sciences.

Mitochondrial F-type ATP synthase: multiple enzyme functions revealed by the membrane-embedded FO structure

Cristina Algieri, F. Trombetti, S. Nesci, 2020, Critical reviews in biochemistry and molecular biology.

Effects of cryopreservation on the mitochondrial bioenergetics of bovine sperm

M. Yeste, D. Bucci, Cristina Algieri, 2022, Reproduction in domestic animals = Zuchthygiene.

Sperm function and mitochondrial activity: An insight on boar sperm metabolism.

G. Galeati, D. Bucci, Cristina Algieri, 2020, Theriogenology.

Modulation of the F1FO-ATPase function by butyltin compounds

S. Nesci, A. Pagliarani, V. Ventrella, 2013 .

Structural and functional changes in gill mitochondrial membranes from the Mediterranean mussel Mytilus galloprovincialis exposed to tri‐n‐butyltin

R. Fiorini, S. Nesci, A. Pagliarani, 2012, Environmental toxicology and chemistry.

Tributyltin-driven enhancement of the DCCD insensitive Mg-ATPase activity in mussel digestive gland mitochondria.

F. Trombetti, S. Nesci, A. Pagliarani, 2012, Biochimie.

A Lethal Channel between the ATP Synthase Monomers.

S. Nesci, Salvatore Nesci, 2018, Trends in biochemical sciences.

Mitochondrial permeability transition, F1FO‐ATPase and calcium: an enigmatic triangle

S. Nesci, Salvatore Nesci, 2017, EMBO reports.