Matthew Alsaloum

Contributions of NaV1.8 and NaV1.9 to excitability in human induced pluripotent stem-cell derived somatosensory neurons

S. Waxman, M. Estacion, F. Dib-Hajj, 2021, Scientific Reports.

Rat NaV1.7 loss-of-function genetic model: Deficient nociceptive and neuropathic pain behavior with retained olfactory function and intra-epidermal nerve fibers

S. Waxman, B. Moyer, Dong Liu, 2019, Molecular pain.

A gain-of-function sodium channel β2-subunit mutation in painful diabetic neuropathy

S. Dib-Hajj, S. Waxman, G. Lauria, 2019, Molecular pain.

The impact of topical treatment for chalazia on the odds of procedural management.

Jill C. Rotruck, Peter Alsaloum, Matthew Alsaloum, 2023, Journal of AAPOS : the official publication of the American Association for Pediatric Ophthalmology and Strabismus.

A Molecular Determinant of Subtype-Specific Desensitization in Ionotropic Glutamate Receptors

L. Wollmuth, R. Kazi, Quan Gan, 2016, The Journal of Neuroscience.

Lacosamide Inhibition of NaV1.7 Channels Depends on its Interaction With the Voltage Sensor Domain and the Channel Pore

H. Smeets, S. Dib-Hajj, S. Waxman, 2021, Frontiers in Pharmacology.

iPSCs and DRGs: stepping stones to new pain therapies.

S. Waxman, M. Alsaloum, Matthew Alsaloum, 2021, Trends in molecular medicine.

Paclitaxel increases axonal localization and vesicular trafficking of Nav1.7

S. Dib-Hajj, S. Waxman, F. Dib-Hajj, 2021, Brain : a journal of neurology.

Status of peripheral sodium channel blockers for non-addictive pain treatment

S. Waxman, Philip R. Effraim, Grant P. Higerd, 2020, Nature Reviews Neurology.