Tom H. Lindner

A novel syndrome of diabetes mellitus, renal dysfunction and genital malformation associated with a partial deletion of the pseudo-POU domain of hepatocyte nuclear factor-1beta.

L. Bostad, P. Njølstad, O. Søvik, 1999, Human molecular genetics.

An Approach for Identifying Simple Sequence Repeat DNA Polymorphisms Near Cloned cDNAs and Genes: Linkage Studies of the Islet Amyloid Polypeptide/Amylin and Liver Glycogen Synthase Genes and NIDDM

R. Spielman, G. Bell, C. Hanis, 1996, Diabetes.

The 31-cM Region of Chromosome 11 Including the Obesity Gene Tubby and ATP-Sensitive Potassium Channel Genes, SUR1 and Kir6.2, Does Not Contain a Major Susceptibility Locus for NIDDM in 127 Non-Hispanic White Affected Sibships

G. Bell, C. Gragnoli, J. Schulze, 1997, Diabetes.

A German genome-wide linkage scan for type 2 diabetes supports the existence of a metabolic syndrome locus on chromosome 1p36.13 and a type 2 diabetes locus on chromosome 16p12.2

T. Wienker, H. Wichmann, Cheryl A. Roe, 2007, Diabetologia.

A genome–wide search for human non–insulin–dependent (type 2) diabetes genes reveals a major susceptibility locus on chromosome 2

R. S. Spielman, G. I. Bell, E. Boerwinkle, 1996, Nature Genetics.

Hepatic function in a family with a nonsense mutation (R154X) in the hepatocyte nuclear factor-4alpha/MODY1 gene.

G. Bell, H. Furuta, B. Cockburn, 1997, The Journal of clinical investigation.