Kenneth S. Gresham

发表

Dose-Dependent Effects of the Myosin Activator Omecamtiv Mecarbil on Cross-Bridge Behavior and Force Generation in Failing Human Myocardium

C. D. dos Remedios, S. Verma, Amy Li, 2017, Circulation. Heart failure.

The contributions of cardiac myosin binding protein C and troponin I phosphorylation to β‐adrenergic enhancement of in vivo cardiac function

J. Stelzer, K. Gresham, Kenneth S. Gresham, 2015, The Journal of physiology.

Cardiac Myosin Binding Protein-C Phosphorylation Modulates Myofilament Length-Dependent Activation

S. Verma, R. Mamidi, J. Stelzer, 2016, Front. Physiol..

Molecular effects of the myosin activator omecamtiv mecarbil on contractile properties of skinned myocardium lacking cardiac myosin binding protein-C.

C. D. dos Remedios, Amy Li, R. Mamidi, 2015, Journal of molecular and cellular cardiology.

Length-dependent changes in contractile dynamics are blunted due to cardiac myosin binding protein-C ablation

R. Mamidi, J. Stelzer, K. Gresham, 2014, Front. Physiol..

The contribution of cardiac myosin binding protein‐c Ser282 phosphorylation to the rate of force generation and in vivo cardiac contractility

R. Mamidi, J. Stelzer, K. Gresham, 2014, The Journal of physiology.

intracellular a cidosis and pH r egulation in Central r espiratory Chemoreceptors

K. Gresham, M. Gdovin, James N. Bayne, 2011 .

Sarcomere-based genetic enhancement of systolic cardiac function in a murine model of dilated cardiomyopathy.

I. Deschênes, X. Wan, R. Mamidi, 2018, International journal of cardiology.

G protein-coupled receptor kinases as therapeutic targets in the heart

W. Koch, K. Gresham, J. Pfleger, 2019, Nature Reviews Cardiology.

MyBP-C Phosphorylation Regulates Ventricular Pressure Development and Relaxation

K. Gresham, Kenneth S. Gresham, 2016 .

Impaired contractile function due to decreased cardiac myosin binding protein C content in the sarcomere.

M. Chandler, Y. Cheng, D. Rosenbaum, 2013, American journal of physiology. Heart and circulatory physiology.

Cardiac myosin binding protein-C Ser302 phosphorylation regulates cardiac β-adrenergic reserve

R. Mamidi, Jiayang Li, J. Stelzer, 2017, Science Advances.

Sarcomeric protein modification during adrenergic stress enhances cross-bridge kinetics and cardiac output.

R. Mamidi, Jiayang Li, J. Stelzer, 2017, Journal of applied physiology.

Alteration of myocardial GRK2 produces a global metabolic phenotype.

W. Koch, K. Drosatos, J. K. Chuprun, 2014, JCI insight.

A peptide of the N terminus of GRK5 attenuates pressure-overload hypertrophy and heart failure

W. Koch, Rajika Roy, J. K. Chuprun, 2021, Science Signaling.

GRK5 is a regulator of fibroblast activation and cardiac fibrosis

W. Koch, E. Gao, J. K. Chuprun, 2021, Proceedings of the National Academy of Sciences.

Airway inflammation and central respiratory control: Results from in vivo and in vitro neonatal rat

Christopher G. Wilson, Ryan M Foglyano, Richard J Martin, 2011, Respiratory Physiology & Neurobiology.

Alteration of myocardial GRK2 produces a global metabolic phenotype.

J. K. Chuprun, A. Cannavo, B. Woodall, 2019, JCI insight.

The contribution of cMyBP-C Ser 282 phosphorylation to the rate of force generation and in vivo cardiac contractility

R. Mamidi, J. Stelzer, K. Gresham, 2014 .

Cardiac myosin binding protein-C: a novel sarcomeric target for gene therapy

R. Mamidi, Jiayang Li, J. Stelzer, 2014, Pflügers Archiv - European Journal of Physiology.