Kazuto Matsunaga

Alveolar macrophage cell line MH-S is valuable as an in vitro model for Legionella pneumophila infection.

T. Klein, Yoshimasa Yamamoto, H. Friedman, 2001, American journal of respiratory cell and molecular biology.

Involvement of Nicotinic Acetylcholine Receptors in Suppression of Antimicrobial Activity and Cytokine Responses of Alveolar Macrophages to Legionella pneumophila Infection by Nicotine1

T. Klein, Yoshimasa Yamamoto, H. Friedman, 2001, The Journal of Immunology.

Epigallocatechin Gallate, a Potential Immunomodulatory Agent of Tea Components, Diminishes Cigarette Smoke Condensate-Induced Suppression of Anti-Legionella pneumophila Activity and Cytokine Responses of Alveolar Macrophages

T. Klein, Yoshimasa Yamamoto, H. Friedman, 2002, Clinical and Vaccine Immunology.

Legionella pneumophila Replication in Macrophages Inhibited by Selective Immunomodulatory Effects on Cytokine Formation by Epigallocatechin Gallate, a Major Form of Tea Catechins

T. Klein, Yoshimasa Yamamoto, H. Friedman, 2001, Infection and Immunity.

In vitro therapeutic effect of epigallocatechin gallate on nicotine-induced impairment of resistance to Legionella pneumophila infection of established MH-S alveolar macrophages.

T. Klein, Yoshimasa Yamamoto, H. Friedman, 2002, The Journal of infectious diseases.

Legionella pneumophila Suppresses Macrophage Interleukin-12 Production by Activating the p42/44 Mitogen-Activated Protein Kinase Cascade

T. Klein, Yoshimasa Yamamoto, H. Friedman, 2003, Infection and Immunity.

Legionella pneumophila Suppresses Interleukin-12 Production by Macrophages

C. Newton, T. Klein, Yoshimasa Yamamoto, 2001, Infection and Immunity.