Howard L. Weiner

Suppression of insulitis in non-obese diabetic (NOD) mice by oral insulin administration is associated with selective expression of interleukin-4 and -10, transforming growth factor-beta, and prostaglandin-E.

H. Weiner, M. Polanski, W. Hancock, 1995, The American journal of pathology.

Oral, but not intravenous, alloantigen prevents accelerated allograft rejection by selective intragraft Th2 cell activation.

H. Weiner, M. Sayegh, W. Hancock, 1993, Transplantation.

Mechanisms of acquired thymic tolerance in experimental autoimmune encephalomyelitis: thymic dendritic-enriched cells induce specific peripheral T cell unresponsiveness in vivo

Howard L. Weiner, Lorenzo Gallon, Wayne W. Hancock, 1995, The Journal of experimental medicine.

Low-Frequency and Rare-Coding Variation Contributes to Multiple Sclerosis Risk

H. Ullum, T. Werge, H. Hakonarson, 2020, Cell.

Acquired tolerance to experimental autoimmune encephalomyelitis by intrathymic injection of myelin basic protein or its major encephalitogenic peptide

H. Weiner, S. Khoury, M. Sayegh, 1993, The Journal of experimental medicine.

Identification of genetic loci associated with paralysis, inflammation and weight loss in mouse experimental autoimmune encephalomyelitis.

J. Seidman, H. Weiner, V. Kuchroo, 2001, International immunology.

Genetic analysis of susceptibility to experimental autoimmune encephalomyelitis in a cross between SJL/J and B10.S mice.

J. Seidman, H. Weiner, V. Kuchroo, 1996, Journal of immunology.

Inheritance of susceptibility to experimental autoimmune encephalomyelitis

Howard L. Weiner, Jeffrey A. Encinas, Vijay K. Kuchroo, 1996, Journal of neuroscience research.