A. Malcolm R. Taylor

p53 dysfunction in B-cell chronic lymphocytic leukemia: inactivation of ATM as an alternative to TP53 mutation.

A. Pettitt, T. Stankovic, G. Stewart, 2001, Blood.

Apoptotic resistance to ionizing radiation in pediatric B-precursor acute lymphoblastic leukemia frequently involves increased NF-kappaB survival pathway signaling.

T. Stankovic, P. Moss, A. Taylor, 2004, Blood.

Microarray analysis reveals that TP53- and ATM-mutant B-CLLs share a defect in activating proapoptotic responses after DNA damage but are distinguished by major differences in activating prosurvival responses.

T. Stankovic, G. Stewart, P. Byrd, 2004, Blood.

A novel CDK inhibitor, CYC202 (R-roscovitine), overcomes the defect in p53-dependent apoptosis in B-CLL by down-regulation of genes involved in transcription regulation and survival.

David P Lane, T. Stankovic, C. Fegan, 2005, Blood.

Ataxia telangiectasia mutated-deficient B-cell chronic lymphocytic leukemia occurs in pregerminal center cells and results in defective damage response and unrepaired chromosome damage.

P. Biggs, Grant S. Stewart, Philip J. Byrd, 2002, Blood.

Bmi-1 is induced by the Epstein-Barr virus oncogene LMP1 and regulates the expression of viral target genes in Hodgkin lymphoma cells.

A. Taylor, C. Woodman, L. Young, 2007, Blood.