Peter Herrlich

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Heterodimer formation of cJun and ATF‐2 is responsible for induction of c‐jun by the 243 amino acid adenovirus E1A protein.

P. Herrlich, A. Zantema, A. Eb, 1993, The EMBO journal.

Interference between pathway‐specific transcription factors: glucocorticoids antagonize phorbol ester‐induced AP‐1 activity without altering AP‐1 site occupation in vivo.

P. Herrlich, H. Rahmsdorf, H. König, 1992, The EMBO journal.

Activation of the c-fos gene by UV and phorbol ester: different signal transduction pathways converge to the same enhancer element.

M. Karin, P. Herrlich, H. Rahmsdorf, 1988, Oncogene.

Induction of metallothionein and other mRNA species by carcinogens and tumor promoters in primary human skin fibroblasts

P. Herrlich, P. Angel, H. Rahmsdorf, 1986, Molecular and cellular biology.

UV‐induced activation of AP‐1 involves obligatory extranuclear steps including Raf‐1 kinase.

P. Herrlich, S. Gebel, P. Angel, 1993, The EMBO journal.

The mammalian UV response: mechanism of DNA damage induced gene expression.

P. Herrlich, S. Gebel, C. Blattner, 1994, Advances in enzyme regulation.

A splice variant of CD44 expressed in the apical ectodermal ridge presents fibroblast growth factors to limb mesenchyme and is required for limb outgrowth.

P. Herrlich, H Ponta, P Herrlich, 1998, Genes & development.

Trans‐acting factors regulate the expression of CD44 splice variants.

P. Herrlich, J. Moll, H. König, 1996, The EMBO journal.

How tumor cells make use of CD44.

J. Sleeman, P. Herrlich, F. Hilberg, 1998, Cell adhesion and communication.

Subfragments of the large terminal repeat cause glucocorticoid-responsive expression of mouse mammary tumor virus and of an adjacent gene.

B. Groner, P. Herrlich, N. Hynes, 1983, Proceedings of the National Academy of Sciences of the United States of America.

Interference of pathway specific transcription factors.

P. Herrlich, H. Ponta, A. Cato, 1992, Biochimica et biophysica acta.

DNA damage-induced gene expression: signal transduction and relation to growth factor signaling.

P. Herrlich, H. Rahmsdorf, H. Ponta, 1992, Reviews of physiology, biochemistry and pharmacology.

UV irradiation-induced interleukin-1 and basic fibroblast growth factor synthesis and release mediate part of the UV response.

P. Herrlich, H. Rahmsdorf, C. Sachsenmaier, 1993, The Journal of biological chemistry.

UV-induced signal transduction.

P. Herrlich, M. Iordanov, C. Blattner, 1997, Journal of photochemistry and photobiology. B, Biology.

Posttranscriptional regulation of c-fos mRNA expression.

A. Schönthal, P. Herrlich, P. Angel, 1987, Nucleic acids research.

Protein kinase induction in Escherichia coli by bacteriophage T7.

F. Studier, P. Herrlich, R. Roskoski, 1974, Proceedings of the National Academy of Sciences of the United States of America.

Radiation-induced activation of transcription factors in mammalian cells

P. Herrlich, H. Grunicke, H. Loferer, 1990, Radiation and environmental biophysics.

Autoregulation of fos: the dyad symmetry element as the major target of repression.

A. Schönthal, P. Herrlich, H. Rahmsdorf, 1989, The EMBO journal.

Both ATF-2 and c-Jun are phosphorylated by stress-activated protein kinases in response to UV irradiation.

I. Herr, P. Herrlich, H. van Dam, 1995, Immunobiology.

The Fos and Jun/AP-1 proteins are involved in the downregulation of Fos transcription.

M. Karin, A. Schönthal, P. Herrlich, 1989, Oncogene.