Andrew J. Saxon

Enhanced human IgE production results from exposure to the aromatic hydrocarbons from diesel exhaust: direct effects on B-cell IgE production.

A. Saxon, D. Díaz-Sánchez, A. Tsien, 1995, The Journal of allergy and clinical immunology.

Enhanced nasal cytokine production in human beings after in vivo challenge with diesel exhaust particles.

A. Saxon, D. Díaz-Sánchez, A. Casillas, 1996, The Journal of allergy and clinical immunology.

Multicenter, double-blind, placebo-controlled, multiple-challenge evaluation of reported reactions to monosodium glutamate.

P. Yarnold, R. Geha, A. Saxon, 2000, The Journal of allergy and clinical immunology.

The organic component of diesel exhaust particles and phenanthrene, a major polyaromatic hydrocarbon constituent, enhances IgE production by IgE-secreting EBV-transformed human B cells in vitro.

A. Saxon, D. Díaz-Sánchez, A. Tsien, 1997, Toxicology and applied pharmacology.

Early IL-4 production driving Th2 differentiation in a human in vivo allergic model is mast cell derived.

A. Saxon, D. Díaz-Sánchez, M. Wang, 1999, Clinical immunology.

In vivo nasal challenge with diesel exhaust particles enhances expression of the CC chemokines rantes, MIP-1alpha, and MCP-3 in humans.

A. Nel, A. Saxon, D. Díaz-Sánchez, 2000, Clinical immunology.

Macrophage activation by polycyclic aromatic hydrocarbons: evidence for the involvement of stress-activated protein kinases, activator protein-1, and antioxidant response elements.

A. Nel, A. Saxon, T. Hiura, 1998, Journal of immunology.