S J Parsons

pp60c-src tyrosine kinase, myristylation, and modulatory domains are required for enhanced mitogenic responsiveness to epidermal growth factor seen in cells overexpressing c-src

J. Parsons, S. Parsons, D. Luttrell, 1989, Molecular and cellular biology.

Augmented mitogenic responsiveness to epidermal growth factor in murine fibroblasts that overexpress pp60c-src

L. Luttrell, S. Parsons, D. Luttrell, 1988, Molecular and cellular biology.

Tyrosine phosphorylation of protein phosphatase 2A in response to growth stimulation and v-src transformation of fibroblasts.

J. Chen, D. Brautigan, Jing Chen, 1994, The Journal of biological chemistry.

Role of c-Src tyrosine kinase in EGF-induced mitogenesis.

S. Parsons, A. Belsches, M. D. Haskell, 1997, Frontiers in bioscience : a journal and virtual library.

c-Src regulates the simultaneous rearrangement of actin cytoskeleton, p190RhoGAP, and p120RasGAP following epidermal growth factor stimulation

J. Settleman, J. Chang, S. Parsons, 1995, The Journal of cell biology.

Increased levels of p21ras-GTP and enhanced DNA synthesis accompany elevated tyrosyl phosphorylation of GAP-associated proteins, p190 and p62, in c-src overexpressors.

J. Chang, S. Parsons, J. Moyers, 1993, Oncogene.

Identification of the in vitro phosphorylation sites on Gs alpha mediated by pp60c-src.

S. Parsons, M. Linder, J. Moyers, 1995, The Biochemical journal.

Androgen receptor degradation by the E3 ligase CHIP modulates mitotic arrest in prostate cancer cells

J. Larner, D. Brautigan, S. Parsons, 2014, Oncogene.

A 42-kD tyrosine kinase substrate linked to chromaffin cell secretion exhibits an associated MAP kinase activity and is highly related to a 42-kD mitogen-stimulated protein in fibroblasts

S. Kanner, T. Sturgill, J. Litz, 1990, The Journal of cell biology.