Alessandro Sette

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Relative contribution of "determinant selection" and "holes in the T-cell repertoire" to T-cell responses.

M. Bekoff, H. Grey, A. Sette, 1989, Proceedings of the National Academy of Sciences of the United States of America.

Effect of pH on MHC class II-peptide interactions.

J. Sidney, H. Grey, A. Sette, 1992, Journal of immunology.

Prediction of major histocompatibility complex binding regions of protein antigens by sequence pattern analysis.

H. Grey, A. Sette, S. Buus, 1989, Proceedings of the National Academy of Sciences of the United States of America.

Definition of specific peptide motifs for four major HLA-A alleles.

A Sette, H. Grey, A. Sette, 1994, Journal of immunology.

A T cell receptor antagonist peptide induces T cells that mediate bystander suppression and prevent autoimmune encephalomyelitis induced with multiple myelin antigens.

A. Sette, B. Hafler, L. Nicholson, 1997, Proceedings of the National Academy of Sciences of the United States of America.

Effect of T-cell receptor antagonism on interaction between T cells and antigen-presenting cells and on T-cell signaling events.

H. Grey, A. Sette, J. Alexander, 1993, Proceedings of the National Academy of Sciences of the United States of America.

Autologous peptides constitutively occupy the antigen binding site on Ia.

H. Grey, A. Sette, S. Buus, 1988, Science.

I-Ad-binding peptides derived from unrelated protein antigens share a common structural motif.

H. Grey, A. Sette, S. Buus, 1988, Journal of immunology.

Recognition of multiple peptide cores by a single T cell receptor

A. Sette, E. Sercarz, H. Geysen, 1995, The Journal of experimental medicine.

The minimal number of class II MHC-antigen complexes needed for T cell activation.

H. Grey, A. Sette, S. Demotz, 1990, Science.

Inhibition of experimental autoimmune encephalomyelitis induction in SJL/J mice by using a peptide with high affinity for IAs molecules.

H. Grey, A. Sette, S. Colon, 1990, Journal of Immunology.

Complementary mutations in an antigenic peptide allow for crossreactivity of autoreactive T-cell clones.

A Sette, A. Sette, D. Hafler, 1996, Proceedings of the National Academy of Sciences of the United States of America.

A disease-related epitope of Torpedo acetylcholine receptor. Residues involved in I-Ab binding, self-nonself discrimination, and TCR antagonism.

K. A. Wall, A. Sette, S. Southwood, 1994, Journal of immunology.

Invariant chain peptides in most HLA-DR molecules of an antigen-processing mutant.

S. Ceman, A. Sette, H. Michel, 1992, Science.

Characterization of a naturally processed MHC class II-restricted T-cell determinant of hen egg lysozyme

H. Grey, A. Sette, E. Appella, 1989, Nature.

Negative selection of CD4+ CD8+ thymocytes by T-cell receptor peptide antagonists.

H. Grey, A. Sette, E. Appella, 1994, Proceedings of the National Academy of Sciences of the United States of America.

HLA-DR3 molecules can bind peptides carrying two alternative specific submotifs.

C Oseroff, A Sette, A. Sette, 1994, Journal of immunology.

Epitope selection and development of peptide based vaccines to treat cancer.

H. Grey, A. Sette, E. Celis, 1995, Seminars in cancer biology.

This information is current as Carcinomas and Melanomas CTL Against Autologous and Allogeneic Peptide Epitopes That Can Elicit Specific Identification of New HER2/neu-Derived

J. Charo, C. Hising, M. Petersson, 2013 .

MHC interaction and T cell recognition of carbohydrates and glycopeptides.

H. Grey, A. Sette, F. Gaeta, 1992, Journal of immunology.