John M. Harlan

Lipid IVA inhibits synthesis and release of tumor necrosis factor induced by lipopolysaccharide in human whole blood ex vivo

C. Raetz, R. Munford, J. Harlan, 1990, The Journal of experimental medicine.

An endothelial cell surface factor(s) induced in vitro by lipopolysaccharide, interleukin 1, and tumor necrosis factor-alpha increases neutrophil adherence by a CDw18-dependent mechanism.

H. Ochs, P. Beatty, K. Stanness, 1986, Journal of immunology.

Lipopolysaccharide-mediated bovine endothelial cell injury in vitro.

G. Striker, S. Schwartz, C. Gajdusek, 1983, Laboratory investigation; a journal of technical methods and pathology.

Cloning of human Bcl-2 homologue: inflammatory cytokines induce human A1 in cultured endothelial cells.

A. Karsan, K. Kaushansky, J. Harlan, 1996, Blood.

A novel beta 1 integrin-dependent mechanism of leukocyte adherence to apoptotic cells.

A. Karsan, J. Harlan, B. Schwartz, 1999, Journal of immunology.

Activation of endothelial-leukocyte adhesion molecule 1 (ELAM-1) gene transcription.

P. Tarr, R. Tizard, T. Pohlman, 1991, Proceedings of the National Academy of Sciences of the United States of America.

Human endothelial cell response to lipopolysaccharide, interleukin-1, and tumor necrosis factor is regulated by protein synthesis.

T. Pohlman, J. Harlan, John M. Harlan, 1989, Cellular immunology.

Deacylated lipopolysaccharide inhibits neutrophil adherence to endothelium induced by lipopolysaccharide in vitro

J. Reisch, K. Chien, W. Campbell, 1987, The Journal of experimental medicine.