P. G. Ince

发表

Amyotrophic Lateral Sclerosis Associated with Genetic Abnormalities in the Gene Encoding Cu/Zn Superoxide Dismutase: Molecular Pathology of Five New Cases, and Comparison with Previous Reports and 73 Sporadic Cases of ALS

J. Slade, P. Ince, P. Shaw, 1998, Journal of neuropathology and experimental neurology.

CSF and plasma amino acid levels in motor neuron disease: elevation of CSF glutamate in a subset of patients.

P. Ince, P. Shaw, J. Richardson, 1995, Neurodegeneration : a journal for neurodegenerative disorders, neuroprotection, and neuroregeneration.

Molecular pathology and genetic advances in amyotrophic lateral sclerosis: an emerging molecular pathway and the significance of glial pathology

J. Highley, Hitoshi Takahashi, M. Strong, 2011, Acta Neuropathologica.

Novel FUS/TLS mutations and pathology in familial and sporadic amyotrophic lateral sclerosis.

J. Highley, H. Hollinger, J. Hartley, 2010, Archives of neurology.

CADASIL and genetics of cerebral ischaemia.

C. Morris, J. Slade, R. Kalaria, 2002, Journal of neural transmission. Supplementum.

Population variation in oxidative stress and astrocyte DNA damage in relation to Alzheimer‐type pathology in the ageing brain

C. Brayne, G. Forster, F. Matthews, 2010, Neuropathology and applied neurobiology.

Isolation of enriched glial populations from post-mortem human CNS material by immuno-laser capture microdissection

B. Sharrack, P. Ince, P. Heath, 2012, Journal of Neuroscience Methods.

Parvalbumin and calbindin D‐28k in the human motor system and in motor neuron disease

C. Heizmann, J. Slade, P. Ince, 1993, Neuropathology and applied neurobiology.

Quantitative assessment of AMPA receptor mRNA in human spinal motor neurons isolated by laser capture microdissection

P. Ince, P. Ince, P. Shaw, 2002, Neuroreport.

Broad clinical phenotypes associated with TAR-DNA binding protein (TARDBP) mutations in amyotrophic lateral sclerosis

J. Highley, K. Morrison, E. Goodall, 2010, neurogenetics.

Neutron activation analysis of trace elements in motor neuron disease spinal cord.

W. D. Ehmann, W. Markesbery, P. Ince, 1995, Neurodegeneration : a journal for neurodegenerative disorders, neuroprotection, and neuroregeneration.

Antisense RNA foci in the motor neurons of C9ORF72-ALS patients are associated with TDP-43 proteinopathy

Paul G. Ince, Stephen B. Wharton, J. Robin Highley, 2015, Acta Neuropathologica.

The expression of the glutamate re-uptake transporter excitatory amino acid transporter 1 (EAAT1) in the normal human CNS and in motor neurone disease: an immunohistochemical study

P. Ince, P. Shaw, S. Banner, 2002, Neuroscience.

The expression of voltage-dependent calcium channel beta subunits in human hippocampus.

P. Ince, P. Shaw, W. Smith, 1998, Brain research. Molecular brain research.

Clinico-pathological features in amyotrophic lateral sclerosis with expansions in C9ORF72.

J. Hardy, B. Traynor, J. Highley, 2012, Brain : a journal of neurology.

Expression of the glial glutamate transporter EAAT2 in the human CNS: an immunohistochemical study.

P. Ince, P. Shaw, C. Horne, 1997, Brain research. Molecular brain research.