O. Douglas Wangensteen

Intercellular adhesion molecule-I (ICAM-I, CD54) deficiency segregates the unique pathophysiological requirements for generating idiopathic pneumonia syndrome (IPS) versus graft-versus-host disease following allogeneic murine bone marrow transplantation.

I. Y. Haddad, B. Blazar, J. Hermanson, 2001, Biology of blood and marrow transplantation : journal of the American Society for Blood and Marrow Transplantation.

KGF pretreatment decreases B7 and granzyme B expression and hastens repair in lungs of mice after allogeneic BMT.

D. Lacey, I. Y. Haddad, P. Bitterman, 2000, American journal of physiology. Lung cellular and molecular physiology.

Acceleration of idiopathic pneumonia syndrome (IPS) in the absence of donor MIP-1 alpha (CCL3) after allogeneic BMT in mice.

J. Serody, A. Panoskaltsis-Mortari, B. Blazar, 2003, Blood.