C. Schneider

Early Presynaptic and Postsynaptic Calcium Signaling Abnormalities Mask Underlying Synaptic Depression in Presymptomatic Alzheimer's Disease Mice

J. Molgó, Grace E. Stutzmann, Shreaya Chakroborty, 2012, The Journal of Neuroscience.

Nitric Oxide Signaling Is Recruited As a Compensatory Mechanism for Sustaining Synaptic Plasticity in Alzheimer's Disease Mice

A. West, Grace E. Stutzmann, Shreaya Chakroborty, 2015, The Journal of Neuroscience.

Reduced presynaptic vesicle stores mediate cellular and network plasticity defects in an early-stage mouse model of Alzheimer’s disease

A. West, D. Peterson, W. Frost, 2019, Molecular Neurodegeneration.

Beta amyloid peptide plaques fail to alter evoked neuronal calcium signals in APP/PS1 Alzheimer's disease mice

J. Richardson, C. Briggs, Grace E. Stutzmann, 2013, Neurobiology of Aging.

Stabilizing ER Ca2+ Channel Function as an Early Preventative Strategy for Alzheimer’s Disease

J. Richardson, C. Briggs, Ivan V. Goussakov, 2012, PloS one.

Early Presynaptic and Postsynaptic Calcium Signaling Abnormalities Mask Underlying Synaptic Depression in Presymptomatic Alzheimer's Disease Mice

J. Molgó, Grace E. Stutzmann, Shreaya Chakroborty, 2012, The Journal of Neuroscience.

Stabilizing ER Ca 2 + Channel Function as an Early Preventative Strategy for Alzheimer ’ s Disease

J. Richardson, C. Briggs, Ivan V. Goussakov, 2012 .