Fletcher B. Taylor

Effect of C1 inhibitor on inflammatory and physiologic response patterns in primates suffering from lethal septic shock.

C. Hack, B. Eisele, P. Jansen, 1998, Journal of immunology.

Survival of primates in LD100 septic shock following therapy with antibody to tumor necrosis factor (TNF alpha).

A. Chang, G. Peer, T. Emerson, 1990, Circulatory shock.

Characterization of the baboon responses to Shiga-like toxin: descriptive study of a new primate model of toxic responses to Stx-1.

T. Pysher, F. Taylor, A. Chang, 1999, The American journal of pathology.

Baboon model of Escherichia coli sepsis: description of its four stages and the role of tumor necrosis factor, tissue factors, and the protein C system in septic shock.

F. Taylor, A. Chang, L. Hinshaw, 1991, Current studies in hematology and blood transfusion.

Staging of the baboon response to group A streptococci administered intramuscularly: a descriptive study of the clinical symptoms and clinical chemical response patterns.

A. Bryant, Dennis L. Stevens, Stanley D. Kosanke, 1999, Clinical infectious diseases : an official publication of the Infectious Diseases Society of America.

Inhibition of factor XII in septic baboons attenuates the activation of complement and fibrinolytic systems and reduces the release of interleukin-6 and neutrophil elastase.

R. Colman, C. Hack, P. Jansen, 1996, Blood.

Activation patterns of coagulation and fibrinolysis in baboons following infusion with lethal or sublethal dose of Escherichia coli.

C. Hack, A. Creasy, A. Eerenberg, 1993, Circulatory shock.