Sayer Al-harbi

Acidosis Sensing Receptor GPR65 Correlates with Anti-Apoptotic Bcl-2 Family Member Expression in CLL Cells: Potential Implications for the CLL Microenvironment

A. Sattar, A. Almasan, B. Hill, 2014, Journal of leukemia.

An update on the molecular pathogenesis and potential therapeutic targeting of AML with t(8;21)(q22;q22.1);RUNX1-RUNX1T1.

M. Mohty, M. Aljurf, S. Ahmed, 2020, Blood advances.

Caspase-3 activation is a critical determinant of genotoxic stress-induced apoptosis.

A. Almasan, G. Choudhary, S. Al-harbi, 2015, Methods in molecular biology.

MCL-1 and BCL-xL-dependent resistance to the BCL-2 inhibitor ABT-199 can be overcome by preventing PI3K/AKT/mTOR activation in lymphoid malignancies

S. Mazumder, A. Almasan, E. Hsi, 2015, Cell Death and Disease.

An antiapoptotic BCL-2 family expression index predicts the response of chronic lymphocytic leukemia to ABT-737.

J. Maciejewski, Kamini Singh, J. Houghton, 2011, Blood.

Clinicopathological analysis of papillary thyroid cancer with PIK3CA alterations in a Middle Eastern population.

Z. Jehan, K. Al-Kuraya, J. Abubaker, 2008, The Journal of clinical endocrinology and metabolism.

Clinicopathological analysis of colorectal cancers with PIK3CA mutations in Middle Eastern population

J. Abubaker, S. Uddin, F. Al-Dayel, 2007, Oncogene.

Prognostic significance of alterations in KRAS isoforms KRAS‐4A/4B and KRAS mutations in colorectal carcinoma

K. Al-Kuraya, J. Abubaker, S. Uddin, 2009, The Journal of pathology.

Mcl-1 Phosphorylation defines ABT-737 resistance that can be overcome by increased NOXA expression in leukemic B cells.

A. Almasan, G. Choudhary, S. Al-harbi, 2012, Cancer research.