Yasushi Saito

Lipoprotein lipase mass and activity in severe hypertriglyceridemia.

K. Shirai, J. Kobayashi, Y. Saito, 1993, Clinica chimica acta; international journal of clinical chemistry.

In vivo effects of cis-5,8,11,14,17-20:5 (n-3) and cis-4,7,10,13,16,19-22:6(n-3) on serum lipoproteins, platelet aggregation, and lipid metabolism in the aorta of rats.

M. Shinomiya, N. Morisaki, Y. Saito, 1983, The Tohoku journal of experimental medicine.

The phospholipase-A2 reaction leads to increased monocyte adhesion of endothelial cells via the expression of adhesion molecules.

K. Yokote, N. Morisaki, Y. Saito, 1993, European journal of biochemistry.

Type I hyperlipoproteinemia caused by lipoprotein lipase defect in lipid-interface recognition was relieved by administration of medium-chain triglyceride.

H. Inadera, K. Shirai, J. Kobayashi, 1992, Metabolism: clinical and experimental.

Frequency and role of apo E phenotype in familial hypercholesterolemia and non-familial hyperlipidemia in the Japanese.

M. Shinomiya, K. Shirai, M. Kitahara, 1990, Atherosclerosis.

A heterozygous mutation (the codon for Ser447----a stop codon) in lipoprotein lipase contributes to a defect in lipid interface recognition in a case with type I hyperlipidemia.

K. Shirai, J. Kobayashi, M. Schotz, 1992, Biochemical and biophysical research communications.

Effect of apolipoprotein E3/4 phenotype on postprandial triglycerides and retinyl palmitate metabolism in plasma from hyperlipidemic subjects in Japan.

J. Kobayashi, N. Morisaki, H. Bujo, 2001, Atherosclerosis.

Inhibition of intimal thickening of the carotid artery of rabbits and of outgrowth of explants of aorta by probucol.

M. Shinomiya, K. Shirai, Y. Saito, 1992, Atherosclerosis.