Kazushige Ota

High Fat Diet Triggers a Reduction in Body Fat Mass in Female Mice Deficient for Utx demethylase

H. Okada, T. Ueda, K. Ge, 2019, Scientific Reports.

KDM4B promotes acute myeloid leukemia associated with AML1‐ETO by regulating chromatin accessibility

M. Kawazu, T. Ueda, A. Komuro, 2021, FASEB BioAdvances.

Identification of senescence-associated genes and their networks under oxidative stress by the analysis of Bach1.

S. Ito, K. Igarashi, A. Nakanome, 2011, Antioxidants & redox signaling.

The H3K27 demethylase, Utx, regulates adipogenesis in a differentiation stage-dependent manner

S. Tomida, K. Nishio, H. Okada, 2017, PloS one.

JMJD2B/KDM4B inactivation in adipose tissues accelerates obesity and systemic metabolic abnormalities

H. Okada, M. Kawazu, T. Ueda, 2018, Genes to cells : devoted to molecular & cellular mechanisms.

Bach1 is critical for the transformation of mouse embryonic fibroblasts by RasV12 and maintains ERK signaling

K. Nakayama, T. Kobayashi, M. Ono, 2013, Oncogene.

Bach1 Deficiency and Accompanying Overexpression of Heme Oxygenase-1 Do Not Influence Aging or Tumorigenesis in Mice

K. Igarashi, Jiying Sun, Ari Itoh-Nakadai, 2014, Oxidative medicine and cellular longevity.

Bach1 inhibits oxidative stress–induced cellular senescence by impeding p53 function on chromatin

T. Noda, Y. Katoh, Minoru Yoshida, 2008, Nature Structural &Molecular Biology.

Bach1‐mediated suppression of p53 is inhibited by p19ARF independently of MDM2

K. Igarashi, T. Ikura, K. Ota, 2012, Cancer science.