Bruno Calabretta

发表

The SH3 domain contributes to BCR/ABL-dependent leukemogenesis in vivo: role in adhesion, invasion, and homing.

B. Calabretta, P. Salomoni, P. Włodarski, 1998, Blood.

Resistance to apoptosis in CTLL-2 cells constitutively expressing c-Myb is associated with induction of BCL-2 expression and Myb-dependent regulation of bcl-2 promoter activity.

C. Franceschi, B. Calabretta, P. Salomoni, 1997, Proceedings of the National Academy of Sciences of the United States of America.

Inhibition of protooncogene expression by antisense oligodeoxynucleotides: biological and therapeutic implications.

B. Calabretta, Bruno Calabretta, 1991, Cancer research.

Role of the KIT protooncogene in normal and malignant human hematopoiesis.

B. Calabretta, M. Ratajczak, A. Gewirtz, 1992, Proceedings of the National Academy of Sciences of the United States of America.

Granulocytic differentiation of normal hematopoietic precursor cells induced by transcription factor PU.1 correlates with negative regulation of the c-myb promoter.

B. Calabretta, D. Perrotti, T. Bellón, 1997, Blood.

Myb and ets proteins are candidate regulators of c-kit expression in human hematopoietic cells.

B. Calabretta, M. Ratajczak, A. Gewirtz, 1998, Blood.

A c-myb antisense oligodeoxynucleotide inhibits normal human hematopoiesis in vitro.

B. Calabretta, A. Gewirtz, Bruno Calabretta, 1988, Science.

Suppression of Philadelphia1 leukemia cell growth in mice by BCR-ABL antisense oligodeoxynucleotide.

P. Iversen, G. Zon, B. Calabretta, 1994, Proceedings of the National Academy of Sciences of the United States of America.

Antisense BCR-ABL oligonucleotides induce apoptosis in the Philadelphia chromosome-positive cell line BV173.

B Calabretta, B. Calabretta, T. Skorski, 1994, Leukemia.

Normal and leukemic hematopoietic cells manifest differential sensitivity to inhibitory effects of c-myb antisense oligodeoxynucleotides: an in vitro study relevant to bone marrow purging.

B. Calabretta, M. Ratajczak, A. Gewirtz, 1991, Proceedings of the National Academy of Sciences of the United States of America.

De novo decorin gene expression suppresses the malignant phenotype in human colon cancer cells.

T Skorski, B Calabretta, B. Calabretta, 1995, Proceedings of the National Academy of Sciences of the United States of America.