Todd A. Pearson

发表

Creation of “Humanized” Mice to Study Human Immunity

D. Greiner, L. Shultz, Todd A. Pearson, 2008, Current protocols in immunology.

A new Hu-PBL model for the study of human islet alloreactivity based on NOD-scid mice bearing a targeted mutation in the IL-2 receptor gamma chain gene.

R. Woodland, B. Woda, M. Atkinson, 2008, Clinical immunology.

Humanized SCID mouse models for biomedical research.

D. Greiner, T. Pearson, L. Shultz, 2008, Current topics in microbiology and immunology.

Humanized NOD/LtSz‐scid IL2 Receptor Common Gamma Chain Knockout Mice in Diabetes Research

D. Greiner, T. Pearson, L. Shultz, 2007, Annals of the New York Academy of Sciences.

Dengue Virus Infection and Virus-Specific HLA-A2 Restricted Immune Responses in Humanized NOD-scid IL2rγnull Mice

A. Rothman, D. Greiner, T. Pearson, 2009, PloS one.

Human peripheral blood leucocyte non‐obese diabetic‐severe combined immunodeficiency interleukin‐2 receptor gamma chain gene mouse model of xenogeneic graft‐versus‐host‐like disease and the role of host major histocompatibility complex

D. Greiner, T. Pearson, L. Shultz, 2009, Clinical and experimental immunology.

Non‐obese diabetic–recombination activating gene‐1 (NOD–Rag 1 null) interleukin (IL)‐2 receptor common gamma chain (IL 2 rγnull) null mice: a radioresistant model for human lymphohaematopoietic engraftment

D. Greiner, T. Pearson, L. Shultz, 2008, Clinical and experimental immunology.

T Cell-Specific siRNA Delivery Suppresses HIV-1 Infection in Humanized Mice

Dale L. Greiner, Sung Wan Kim, S. W. Kim, 2008, Cell.

A new immunodeficient hyperglycaemic mouse model based on the Ins2Akita mutation for analyses of human islet and beta stem and progenitor cell function

J. Lief, R. Bottino, D. Greiner, 2008, Diabetologia.

Islet-Specific CTL Cloned from a Type 1 Diabetes Patient Cause Beta-Cell Destruction after Engraftment into HLA-A2 Transgenic NOD/SCID/IL2RG Null Mice

J. Drijfhout, M. Kester, M. Griffioen, 2012, PloS one.

Humanized Mice for the Study of Type 1 Diabetes and Beta Cell Function

D. Greiner, T. Pearson, L. Shultz, 2008, Annals of the New York Academy of Sciences.

Expanded CD34+ Human Umbilical Cord Blood Cells Generate Multiple Lymphohematopoietic Lineages in NOD-scid IL2rγ null Mice

R. Woodland, B. Woda, D. Greiner, 2008, Experimental biology and medicine.

Dengue virus infection and virus-specific HLA-A2 restricted immune responses in humanized NOD-scid IL2rgammanull mice

A. Rothman, D. Greiner, T. Pearson, 2021 .

The Genetic Basis of Resistance to Transplantation Tolerance Induced by Costimulation Blockade in NOD Mice: a Dissertation

T. Pearson, Todd A. Pearson, 2003 .

Rapid quantification of naive alloreactive T cells by TNF-alpha production and correlation with allograft rejection in mice.

D. Greiner, T. Pearson, R. Welsh, 2007, Blood.

Islet Cell Autoimmunity and Transplantation Tolerance: Two Distinct Mechanisms?

L. Wicker, D. Greiner, T. Pearson, 2003, Annals of the New York Academy of Sciences.

TLR Agonists Prevent the Establishment of Allogeneic Hematopoietic Chimerism in Mice Treated with Costimulation Blockade12

M. Yamazaki, D. Greiner, T. Pearson, 2009, The Journal of Immunology.

Treated with Costimulation Blockade Allogeneic Hematopoietic Chimerism in Mice TLR Agonists Prevent the Establishment of

M. Yamazaki, T. Pearson, L. Shultz, 2013 .

Islet allograft survival induced by costimulation blockade in NOD mice is controlled by allelic variants of Idd3.

L. Wicker, D. Greiner, T. Pearson, 2004, Diabetes.

End-Organ Damage in a Mouse Model of Fulminant Liver Inflammation Requires CD4+ T Cell Production of IFN-γ but Is Independent of Fas1

T. Pearson, M. Milks, J. G. Cripps, 2009, The Journal of Immunology.

T Cell Production of IFN-+ CD 4 Fulminant Liver Inflammation Requires End-Organ Damage in a Mouse Model of

T. Pearson, J. G. Cripps, R. Robinson, 2009 .

A Novel Alloantigen-Specific CD8+PD1+ Regulatory T Cell Induced by ICOS-B7h Blockade In Vivo1

G. Freeman, R. Abdi, M. Jurewicz, 2007, The Journal of Immunology.

Partial versus Full Allogeneic Hemopoietic Chimerization Is a Preferential Means to Inhibit Type 1 Diabetes as the Latter Induces Generalized Immunosuppression1

D. Greiner, T. Pearson, D. Serreze, 2006, The Journal of Immunology.

Blockade In Vivo Regulatory T Cell Induced by ICOS-B 7 h + PD 1 + A Novel Alloantigen-Specific CD 8 Sayegh and

G. Freeman, R. Abdi, M. Jurewicz, 2007 .

Different Mechanisms Control Peripheral and Central Tolerance in Hematopoietic Chimeric Mice

M. Yamazaki, D. Greiner, T. Pearson, 2007, American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons.

Idd Loci Synergize to Prolong Islet Allograft Survival Induced by Costimulation Blockade in NOD Mice

L. Wicker, D. Greiner, T. Pearson, 2009, Diabetes.

NOD congenic mice genetically protected from autoimmune diabetes remain resistant to transplantation tolerance induction.

L. Wicker, D. Greiner, T. Pearson, 2003, Diabetes.

TLR Agonists Abrogate Co‐stimulation Blockade‐Induced Mixed Chimerism and Transplantation Tolerance

M. Yamazaki, D. Greiner, T. Pearson, 2008, Annals of the New York Academy of Sciences.

Dengue virus infection and virus‐specific immune responses in humanized NOD‐scid IL2rgammanull mice

A. Rothman, D. Greiner, T. Pearson, 2008 .

Genetic Disassociation of Autoimmunity and Resistance to Costimulation Blockade-Induced Transplantation Tolerance in Nonobese Diabetic Mice1

L. Wicker, D. Greiner, T. Pearson, 2003, The Journal of Immunology.