Jacques A. Louis

The IL-4 rapidly produced in BALB/c mice after infection with Leishmania major down-regulates IL-12 receptor beta 2-chain expression on CD4+ T cells resulting in a state of unresponsiveness to IL-12.

C. Parra-López, J. Louis, F. Tacchini-Cottier, 1998, Journal of immunology.

Expansion of gamma delta+ T cells in BALB/c mice infected with Leishmania major is dependent upon Th2-type CD4+ T cells

A. Hayday, H. Macdonald, M. Owen, 1995, Infection and immunity.

Mice from a Genetically Resistant Background Lacking the Interferon Y Receptor Are Susceptible to Infection with Ieishmania Major but Mount a Polarized T Helper Cell 1-type Cd4 + T Cell Response

R. Behin, K. Swihart, K. Hug, 1995 .

The IL-4 Rapidly Produced in BALB/c Mice After Infection with Leishmania major Down-Regulates IL-12 Receptor β2-Chain Expression on CD4+ T Cells Resulting in a State of Unresponsiveness to IL-12

F. Tacchini-Cottier, H. Himmelrich, Carlos Parra-Lopez, 1998, The Journal of Immunology.

Leishmania major infection in BALB/c mice: protection or exacerbation by treatment with different doses of BCG.

J. Louis, I. Müller, M. M. Stefani, 1993, Research in immunology.

Autoimmune response to DNA, red blood cells, and thymocyte antigens in association with polyclonal antibody synthesis during experimental African trypanosomiasis.

J. Louis, P. Lambert, S. Izui, 1979, Journal of immunology.

Mechanism for induction of anti-DNA antibodies by bacterial lipopolysaccharides in mice; II. Correlation between anti-DNA induction and polyclonal antibody formation by various polyclonal B lymphocyte activators.

J. Louis, P. Lambert, S. Izui, 1977, Journal of immunology.

Immunization of BALB/c mice with Helicobacter urease B induces a T helper 2 response absent in Helicobacter infection.

A. Blum, I. Corthésy-Theulaz, P. Launois, 1998, Gastroenterology.