Suruchi Pacharne

发表

Molecular synergy underlies the co-occurrence patterns and phenotype of NPM1-mutant acute myeloid leukemia.

R. Andrews, O. Dovey, A. Bradley, 2017, Blood.

Molecular synergy underlies the co-occurrence patterns and phenotype of NPM 1-mutant 1 acute myeloid leukemia . 2 3

R. Andrews, O. Dovey, A. Bradley, 2017 .

Accelerated Development With Increased Bone Mass and Skeletal Response to Loading Suggest Receptor Activity Modifying Protein-3 as a Bone Anabolic Target

M. Kadmiel, T. Skerry, K. Caron, 2022, Frontiers in Endocrinology.

Research resource: Haploinsufficiency of receptor activity-modifying protein-2 (RAMP2) causes reduced fertility, hyperprolactinemia, skeletal abnormalities, and endocrine dysfunction in mice.

M. Kadmiel, T. Skerry, K. Caron, 2011, Molecular endocrinology.

Hemopoietic-specific Sf3b1-K700E knock-in mice display the splicing defect seen in human MDS but develop anemia without ring sideroblasts

D. Kent, E. Papaemmanuil, N. Bolli, 2016, Leukemia.

SETBP1 overexpression acts in the place of class-defining mutations to drive FLT3-ITD-mutant AML.

Mathias J Friedrich, O. Dovey, R. Rad, 2021, Blood advances.

ACCELERATED SKELETAL DEVELOPMENT IN RECEPTOR ACTIVITY MODIFYING PROTEIN 3 KNOCK-OUT MICE AND NOVEL INSIGHTS INTO CALCITONIN RECEPTOR IN PRIMARY OSTEOBLASTS.

S. Pacharne, Suruchi Pacharne, 2014 .

Research resource: Haploinsufficiency of receptor activity-modifying protein-2 (RAMP2) causes reduced fertility, hyperprolactinemia, skeletal abnormalities, and endocrine dysfunction in mice.

M. Kadmiel, T. Skerry, K. Caron, 2011, Molecular endocrinology.