Shyr-Te Ju

发表

Cytokines and adhesion molecules contribute to the ability of myelin proteolipid protein-specific T cell clones to mediate experimental allergic encephalomyelitis.

V. Kuchroo, R. Sobel, S. Ju, 1993, Journal of immunology.

Activation-induced apoptosis of mature T cells is dependent upon the level of surface TCR but not on the presence of the CD3 zeta ITAM.

S. Ju, C. Terhorst, K. Matsui, 1998, International immunology.

Identification of two NF-kappa B sites in mouse CD95 ligand (Fas ligand) promoter: functional analysis in T cell hybridoma.

S. Ju, A. Marshak‐Rothstein, A. Fine, 1998, Journal of immunology.

Expression of two distinct cytolytic mechanisms among murine CD4 subsets.

S. Ju, M. Dorf, R. DeKruyff, 1990, Journal of immunology.

Inducer T-cell-mediated killing of antigen-presenting cells.

S. Ju, M. Dorf, R. DeKruyff, 1986, Cellular immunology.

Acceleration of lpr lymphoproliferative and autoimmune disease by transgenic protein kinase CK2 alpha.

D. Salant, D. Seldin, S. Ju, 1998, Journal of immunology.

CD4+ T cells reactivated with superantigen are both more sensitive to FasL-mediated killing and express a higher level of FasL.

S. Ju, A. Marshak‐Rothstein, J. Tschopp, 1997, Cellular immunology.

Production of large amounts of antibodies in individual mice.

S. Ju, S. Sato, A. Nisonoff, 1976, Journal of immunology.

Selective activation of helper and cytolytic T-cell functions of L3T4+ clones with either antireceptor antibody or phorbol ester and ionophore.

S. Ju, M. Dorf, R. DeKruyff, 1987, Cellular immunology.

Regulation of T-cell death genes: selective inhibition of FasL- but not Fas-mediated function.

S. Ju, A. Marshak‐Rothstein, D. Sherr, 1996, Cellular immunology.

Characterization of lpr-derived T cell hybridomas: Fas-deficient hybridomas are deathless, growth-arrested, and cytotoxic upon activation.

S. Ju, M. Sy, A. Marshak‐Rothstein, 1996, Cellular immunology.

The cytotoxic process of CD4 Th1 clones.

V. Kuchroo, S. Ju, M. Ozdemirli, 1992, Journal of immunology.