Ronald M. Evans

Normal development and growth of mice carrying a targeted disruption of the alpha 1 retinoic acid receptor gene.

R. Jaenisch, E. Li, R. Evans, 1993, Proceedings of the National Academy of Sciences of the United States of America.

RXR alpha mutant mice establish a genetic basis for vitamin A signaling in heart morphogenesis.

R. Evans, K. Chien, H. Sucov, 1994, Genes & development.

Compartment-selective sensitivity of cardiovascular morphogenesis to combinations of retinoic acid receptor gene mutations.

R. Evans, V. Giguère, H. Sucov, 1997, Circulation research.

Atrial-like phenotype is associated with embryonic ventricular failure in retinoid X receptor alpha -/- mice.

J. Ross, G. Schmid-Schönbein, R. Evans, 1995, Proceedings of the National Academy of Sciences of the United States of America.

RXR alpha deficiency confers genetic susceptibility for aortic sac, conotruncal, atrioventricular cushion, and ventricular muscle defects in mice.

R. Evans, K. Chien, P. Gruber, 1996, The Journal of clinical investigation.

Retinoid-dependent pathways suppress myocardial cell hypertrophy.

R. Evans, K. Chien, H. Sucov, 1995, Proceedings of the National Academy of Sciences of the United States of America.