G. Velasco

Anticancer mechanisms of cannabinoids.

G. Velasco, M. Guzmán, C. Sánchez, 2016, Current oncology.

Anti-tumoral action of cannabinoids on hepatocellular carcinoma: role of AMPK-dependent activation of autophagy

I. Díaz-Laviada, M. Salazar, D. Vara, 2011, Cell Death and Differentiation.

Anti-tumoral action of cannabinoids on hepatocellular carcinoma: role of AMPK-dependent activation of autophagy

G. Velasco, M. Guzmán, G. Guzman, 2011, Cell Death and Differentiation.

Anti-tumoral action of cannabinoids on hepatocellular carcinoma: role of AMPK-dependent activation of autophagy ThisarticlehasbeencorrectedsinceAdvanceOnlinePublicationandacorrigendumisalsoprintedinthisissue

M. Salazar, D. Vara, N. Olea‐Herrero, 2011 .

A pilot clinical study of Δ9-tetrahydrocannabinol in patients with recurrent glioblastoma multiforme

G. Velasco, I. Galve-Roperh, M. Guzmán, 2006, British Journal of Cancer.

Control of hepatic fatty acid oxidation by 5'-AMP-activated protein kinase involves a malonyl-CoA-dependent and a malonyl-CoA-independent mechanism.

G. Velasco, M. Guzmán, M. Geelen, 1997, Archives of biochemistry and biophysics.

The CB1 cannabinoid receptor is coupled to the activation of protein kinase B/Akt.

G. Velasco, T. Gómez del Pulgar, M. Guzmán, 2000, The Biochemical journal.

The orphan G protein-coupled receptor GPR55 promotes cancer cell proliferation via ERK

G. Velasco, M. Guzmán, M. Lorente, 2011, Oncogene.