C. S. Raine

发表

Reactive nitrogen intermediates in human neuropathology: an overview.

A. Casadevall, D. Dickson, C. Brosnan, 1994, Developmental neuroscience.

Homing to central nervous system vasculature by antigen-specific lymphocytes. I. Localization of 14C-labeled cells during acute, chronic, and relapsing experimental allergic encephalomyelitis.

C. Brosnan, A. Cross, C. Raine, 1990, Laboratory investigation; a journal of technical methods and pathology.

Identification of lymphotoxin and tumor necrosis factor in multiple sclerosis lesions.

C. Brosnan, K. Selmaj, B. Cannella, 1991, The Journal of clinical investigation.

Upregulation and coexpression of adhesion molecules correlate with relapsing autoimmune demyelination in the central nervous system

B. Cannella, A. Cross, C. S. Raine, 1990, The Journal of experimental medicine.

T cell deletion in high antigen dose therapy of autoimmune encephalomyelitis.

J. Goverman, M. Lenardo, C. Raine, 1994, Science.

Multiple sclerosis: immune system molecule expression in the central nervous system.

C. Raine, C. S. Raine, 1994, Journal of neuropathology and experimental neurology.

Notch1 signaling plays a role in regulating precursor differentiation during CNS remyelination

C. Brosnan, D. Rowitch, Changhui Ge, 2009, Proceedings of the National Academy of Sciences.

The neuregulin, glial growth factor 2, diminishes autoimmune demyelination and enhances remyelination in a chronic relapsing model for multiple sclerosis.

C. Raine, B. Cannella, D. Lawson, 1998, Proceedings of the National Academy of Sciences of the United States of America.

Multiple sclerosis. Oligodendrocyte survival and proliferation in an active established lesion.

L. Scheinberg, J. Waltz, C. Raine, 1981, Laboratory investigation; a journal of technical methods and pathology.

IgG in brain correlates with clinicopathological damage in HTLV-1 associated neurologic disease

C. Raine, M. Levin, F. Dohan, 2003, Neurology.

Most patients with multiple sclerosis or a clinically isolated demyelinating syndrome should be treated at the time of diagnosis.

F. Barkhof, M. Filippi, H. Weiner, 2006, Archives of neurology.

Adhesion-related molecules in the central nervous system. Upregulation correlates with inflammatory cell influx during relapsing experimental autoimmune encephalomyelitis.

A. Cross, C. Raine, B. Cannella, 1991, Laboratory investigation; a journal of technical methods and pathology.

A pathogenic role for gamma delta T cells in relapsing-remitting experimental allergic encephalomyelitis in the SJL mouse.

C. Brosnan, C. Raine, A. J. Rajan, 1996, Journal of immunology.