Shephali Bhatnagar

发表

Reciprocal Interaction between TRAF6 and Notch Signaling Regulates Adult Myofiber Regeneration upon Injury

S. Kuang, S. Bhatnagar, Ashok Kumar, 2012, Molecular and Cellular Biology.

The TWEAK-Fn14 System: Breaking the Silence of Cytokine-Induced Skeletal Muscle Wasting

S. Bhatnagar, A. Kumar, Shephali Bhatnagar, 2012, Current molecular medicine.

Genetic ablation of TWEAK augments regeneration and post-injury growth of skeletal muscle in mice.

S. Kuang, S. Bhatnagar, Ashok Kumar, 2010, The American journal of pathology.

TWEAK promotes exercise intolerance by decreasing skeletal muscle oxidative phosphorylation capacity

S. Bhatnagar, Ashok Kumar, Y. Ogura, 2013, Skeletal Muscle.

Resveratrol and Estradiol Rapidly Activate MAPK Signaling through Estrogen Receptors α and β in Endothelial Cells*

C. Klinge, K. A. Blankenship, K. E. Risinger, 2005, Journal of Biological Chemistry.

Resveratrol and estradiol rapidly activate MAPK signaling through estrogen receptors alpha and beta in endothelial cells.

C. Klinge, K. A. Blankenship, K. E. Risinger, 2005, The Journal of biological chemistry.

Genomic Profiling of Messenger RNAs and MicroRNAs Reveals Potential Mechanisms of TWEAK-Induced Skeletal Muscle Wasting in Mice

Siva K. Panguluri, John J. McCarthy, Shephali Bhatnagar, 2010, PloS one.

Therapeutic targeting of signaling pathways in muscular dystrophy

S. Bhatnagar, Ashok Kumar, Shephali Bhatnagar, 2010, Journal of Molecular Medicine.

Osteopontin-Stimulated Expression of Matrix Metalloproteinase-9 Causes Cardiomyopathy in the mdx Model of Duchenne Muscular Dystrophy

S. Tyagi, Ashok Kumar, S. Givvimani, 2011, The Journal of Immunology.

Matrix metalloproteinase-9 inhibition ameliorates pathogenesis and improves skeletal muscle regeneration in muscular dystrophy.

S. Bhatnagar, Ashok Kumar, Denys Y. Makonchuk, 2009, Human molecular genetics.

in the mdx Model of Duchenne Muscular Metalloproteinase-9 Causes Cardiomyopathy Osteopontin-Stimulated Expression of Matrix

Ashok Kumar, Shephali Bhatnagar, Natia Qipshidze, 2015 .

Matrix metalloproteinase inhibitor batimastat alleviates pathology and improves skeletal muscle function in dystrophin-deficient mdx mice.

S. Bhatnagar, Ashok Kumar, Ashok Kumar, 2010, The American journal of pathology.

Regulatory circuitry of TWEAK‐Fn14 system and PGC‐1α in skeletal muscle atrophy program

S. Bhatnagar, Ashok Kumar, T. Zheng, 2014, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

TWEAK and TRAF6 regulate skeletal muscle atrophy

S. Bhatnagar, Ashok Kumar, P. Paul, 2012, Current opinion in clinical nutrition and metabolic care.

TWEAK causes myotube atrophy through coordinated activation of ubiquitin‐proteasome system, autophagy, and caspases

Sanjay Kumar Gupta, S. Bhatnagar, Ashok Kumar, 2012, Journal of cellular physiology.

The E3 Ubiquitin Ligase TRAF6 Intercedes in Starvation-Induced Skeletal Muscle Atrophy through Multiple Mechanisms

S. Bhatnagar, Ashok Kumar, Yongwon Choi, 2012, Molecular and Cellular Biology.

The TWEAK–Fn14 system is a critical regulator of denervation-induced skeletal muscle atrophy in mice

S. Bhatnagar, Denys Y. Makonchuk, Ashok Kumar, 2010, The Journal of cell biology.

Targeted ablation of TRAF6 inhibits skeletal muscle wasting in mice

S. Bhatnagar, Yongwon Choi, Ashok Kumar, 2010, The Journal of cell biology.

Increased microRNA-34c abundance in Alzheimer's disease circulating blood plasma

E. Wang, H. Chertkow, S. Bhatnagar, 2014, Front. Mol. Neurosci..

Elevated levels of active matrix metalloproteinase-9 cause hypertrophy in skeletal muscle of normal and dystrophin-deficient mdx mice.

Shephali Bhatnagar, Saurabh Dahiya, S. Kuang, 2011, Human molecular genetics.

Tumor Necrosis Factor-α Regulates Distinct Molecular Pathways and Gene Networks in Cultured Skeletal Muscle Cells

S. Bhatnagar, Ashok Kumar, R. Lundy, 2010, PloS one.

Transforming Growth Factor-β-activated Kinase 1 Is an Essential Regulator of Myogenic Differentiation*

Denys Y. Makonchuk, Ashok Kumar V, Hong Li, 2009, The Journal of Biological Chemistry.