Michela Zago

发表

Aryl hydrocarbon receptor (AhR) attenuation of subchronic cigarette smoke-induced pulmonary neutrophilia is associated with retention of nuclear RelB and suppression of intercellular adhesion molecule-1 (ICAM-1).

Q. Hamid, C. Baglole, D. Eidelman, 2014, Toxicological sciences : an official journal of the Society of Toxicology.

Aryl hydrocarbon receptor deficiency causes the development of chronic obstructive pulmonary disease through the integration of multiple pathogenic mechanisms

Benjamin M. Smith, M. Obeidat, Y. Bossé, 2021, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Low levels of the AhR in chronic obstructive pulmonary disease (COPD)-derived lung cells increases COX-2 protein by altering mRNA stability

Q. Hamid, P. Nair, C. Baglole, 2017, PloS one.

RelB attenuates cigarette smoke extract‐induced apoptosis in association with transcriptional regulation of the aryl hydrocarbon receptor

John H. White, Q. Hamid, C. Baglole, 2017, Free radical biology & medicine.

Aryl hydrocarbon receptor (AhR)-dependent regulation of pulmonary miRNA by chronic cigarette smoke exposure

C. Baglole, J. Matthews, M. Zago, 2017, Scientific Reports.

The aryl hydrocarbon receptor suppresses cigarette-smoke-induced oxidative stress in association with dioxin response element (DRE)-independent regulation of sulfiredoxin 1.

Q. Hamid, P. Nair, C. Baglole, 2015, Free radical biology & medicine.

Decreased expression of the NF-κB family member RelB in lung fibroblasts from Smokers with and without COPD potentiates cigarette smoke-induced COX-2 expression

J. Bourbeau, A. Benedetti, Q. Hamid, 2015, Respiratory Research.

Alterations in the Expression of the NF-κB Family Member RelB as a Novel Marker of Cardiovascular Outcomes during Acute Exacerbations of Chronic Obstructive Pulmonary Disease

J. Bourbeau, C. Baglole, M. Zago, 2014, PloS one.

Aryl hydrocarbon receptor-dependent regulation of miR-196a expression controls lung fibroblast apoptosis but not proliferation.

Qutayba Hamid, Alvin Gomez, Michela Zago, 2014, Toxicology and applied pharmacology.

Aryl Hydrocarbon Receptor-Dependent Retention of Nuclear HuR Suppresses Cigarette Smoke-Induced Cyclooxygenase-2 Expression Independent of DNA-Binding

Q. Hamid, P. Nair, S. Di Marco, 2013, PloS one.

Genetic Ablation of the Aryl Hydrocarbon Receptor Causes Cigarette Smoke-induced Mitochondrial Dysfunction and Apoptosis*

P. Sime, C. Baglole, R. Phipps, 2011, The Journal of Biological Chemistry.

The NF-κB family member RelB regulates microRNA miR-146a to suppress cigarette smoke-induced COX-2 protein expression in lung fibroblasts.

Q. Hamid, C. Baglole, D. Eidelman, 2014, Toxicology letters.

Fluorescent light energy in wound healing: when is a photon something more?

Dennis Dan Corell, Michael R Hamblin, Shannon E. Campbell, 2020, BiOS.

Suppression of Procollagen α1 Type 1 by Long-Term Low-Dose Exposure to 2-Hydroxyethylmethacrylate in Human Gingival Fibroblasts In Vitro

G. Mazzotti, M. Falconi, G. Teti, 2010, International journal of toxicology.

HEMA down-regulates procollagen alpha1 type I in human gingival fibroblasts.

A. Ruggeri, G. Mazzotti, M. Falconi, 2009, Journal of biomedical materials research. Part A.

Expression of procollagen alpha1 type I and tenascin proteins induced by HEMA in human pulp fibroblasts.

A. Ruggeri, G. Mazzotti, M. Falconi, 2008, Toxicology in vitro : an international journal published in association with BIBRA.

Effects of HEMA on type I collagen protein in human gingival fibroblasts

G. Mazzotti, M. Falconi, G. Teti, 2007, Cell Biology and Toxicology.

Fluorescent Light Energy (FLE) Acts on Mitochondrial Physiology Improving Wound Healing

L. Hébert, F. Bassetto, P. Pinton, 2020, Journal of clinical medicine.

Endocrine aryl hydrocarbon receptor signaling is induced by moderate cutaneous exposure to ultraviolet light

John H. White, C. Baglole, D. Goltzman, 2018, Scientific Reports.