P. Pelicci

发表

Shc proteins are phosphorylated and regulated by the v-Src and v-Fps protein-tyrosine kinases.

T. Pawson, P. Pelicci, A. Cheng, 1992, Proceedings of the National Academy of Sciences of the United States of America.

Oligomerization of RAR and AML1 transcription factors as a novel mechanism of oncogenic activation.

S. Minucci, P. Pelicci, M. Lazar, 2000, Molecular cell.

Genomic characterization of asymptomatic CT-detected lung cancers

E. Venturini, G. Pelosi, G. Veronesi, 2011, Oncogene.

Decitabine, differently from DNMT1 silencing, exerts its antiproliferative activity through p21 upregulation in malignant pleural mesothelioma (MPM) cells.

S. Amatori, M. Fanelli, M. R. Rippo, 2009, Lung cancer.

Structure and origin of the acute promyelocytic leukemia myl/RAR alpha cDNA and characterization of its retinoid-binding and transactivation properties.

P. Pelicci, A. Mencarelli, F. Grignani, 1991, Oncogene.

Deconstructing PML‐induced premature senescence

A. Dejean, P. Pelicci, K. Itahana, 2002, The EMBO journal.

A phase I–II study of the histone deacetylase inhibitor valproic acid plus chemoimmunotherapy in patients with advanced melanoma

A. Goldhirsch, S. Minucci, A. Testori, 2009, British Journal of Cancer.

Retinoid receptors in health and disease: co-regulators and the chromatin connection.

S. Minucci, P. Pelicci, P. Pelicci, 1999, Seminars in cell & developmental biology.

Opposite effects of the acute promyelocytic leukemia PML-retinoic acid receptor alpha (RAR alpha) and PLZF-RAR alpha fusion proteins on retinoic acid signalling

C. Peschle, P. Pelicci, U. Testa, 1997, Molecular and cellular biology.

Frequency of RAS and p53 mutations in acute promyelocytic leukemias.

P. Pelicci, A. Neri, F. Lo Coco, 1993, Leukemia & lymphoma.

Epigenetic gene silencing in acute promyelocytic leukemia.

S. Minucci, P. Pelicci, R. Villa, 2004, Biochemical pharmacology.

Inhibition of histone deacetylase activity by trichostatin A modulates gene expression during mouse embryogenesis without apparent toxicity.

P. Pelicci, G. Cossu, U. Borello, 2001, Cancer research.

Histone deacetylase-targeted treatment restores retinoic acid signaling and differentiation in acute myeloid leukemia.

P G Pelicci, S. Minucci, P. Pelicci, 2001, Cancer research.

PML-RARA-associated cooperating mutations belong to a transcriptional network that is deregulated in myeloid leukemias

D. Bernardo, V. Belcastro, L. Riva, 2017, Leukemia.

Multiple monoclonal B cell expansions and c-myc oncogene rearrangements in acquired immune deficiency syndrome-related lymphoproliferative disorders. Implications for lymphomagenesis

P. Pelicci, P. Luciw, C. Basilico, 1986, The Journal of experimental medicine.

A specific monoclonal antibody (PG-B6) detects expression of the BCL-6 protein in germinal center B cells.

S. Pileri, B. Falini, E. Pescarmona, 1995, The American journal of pathology.

Relationship between the c-myb locus and the 6q-chromosomal aberration in leukemias and lymphomas.

S. Smith, P. Pelicci, C. Barletta, 1987, Science.

Human T lymphocytes expressing TCR gamma/delta.

P. Pelicci, E. Ciccone, L. Moretta, 1990, Research in immunology.

MULTIPLE MONOCLONAL B CELL EXPANSIONS AND c-myc ONCOGENE REARRANGEMENTS IN ACQUIRED IMMUNE DEFICIENCY SYNDROME-RELATED LYMPHOPROLIFERATIVE DISORDERS

Riccardo Dalla-Favera, P. Pelicci, Rosemary Wieczorek, 1986 .

Monoclonal antibodies PG-B6a and PG-B6p recognize, respectively, a highly conserved and a formol-resistant epitope on the human BCL-6 protein amino-terminal region.

S. Pileri, H. Stein, L. Pasqualucci, 1996, The American journal of pathology.

Myeloid and megakaryocytic properties of K-562 cell lines.

W. Vainchenker, P. Pelicci, U. Testa, 1983, Cancer research.

ERRATUM: Inhibitors of histone deacetylases induce tumor-selective apoptosis through activation of the death receptor pathway

L. Altucci, S. Minucci, P. Pelicci, 2005, Nature Medicine.

Shc adaptor proteins are key transducers of mitogenic signaling mediated by the G protein‐coupled thrombin receptor.

J. Pouysségur, D. Grall, E. Van Obberghen-Schilling, 1996, The EMBO journal.

Characterization of a recurrent translocation t(2;3)(p15–22;q26) occurring in acute myeloid leukaemia

M. Mancini, M. Rocchi, G. Specchia, 2006, Leukemia.

Arsenic trioxide (ATO) and MEK1 inhibition synergize to induce apoptosis in acute promyelocytic leukemia cells

F. Lo‐Coco, P. Pelicci, P. Lunghi, 2005, Leukemia.

Constitutive degradation of PML/RARalpha through the proteasome pathway mediates retinoic acid resistance.

S. Minucci, P. Pelicci, C. Nervi, 1999, Blood.

From Cell Populations to Molecular Complexes: Multiplexed Multimodal Microscopy to Explore p53-53BP1 Molecular Interaction

M. Faretta, L. Furia, Simone Pelicci, 2024, International journal of molecular sciences.

retroviral gene transfer into purified murine hematopoietic progenitors PML-RAR induces promyelocytic leukemias with high efficiency following

F. Lo‐Coco, S. Minucci, P. Pelicci, 2002 .

Mapping of the molecular determinants involved in the interaction between eps15 and AP-2.

P. Pelicci, G. Carpenter, P. P. Di Fiore, 1997, Cancer research.

Factor V Leiden and G20210A prothrombin mutation and the risk of subclavian vein thrombosis in patients with breast cancer and a central venous catheter.

G. Severi, F. Orsi, A. Goldhirsch, 2004, Annals of oncology : official journal of the European Society for Medical Oncology.

NPMc+ and FLT3_ITD mutations cooperate in inducing acute leukaemia in a novel mouse model

G. Pruneri, A. Viale, E. Colombo, 2013, Leukemia.

A phase I–II study of the histone deacetylase inhibitor valproic acid plus chemoimmunotherapy in patients with advanced melanoma

A. Goldhirsch, S. Minucci, A. Rocca, 2009, British Journal of Cancer.

Detection of homozygous deletions of the cyclin-dependent kinase 4 inhibitor (p16) gene in acute lymphoblastic leukemia and association with adverse prognostic features.

F. Mandelli, P. Pelicci, F. Lo Coco, 1995, Blood.