Naoki Nakashima

Effects of General Receptor for Phosphoinositides 1 on Insulin and Insulin-Like Growth Factor I-Induced Cytoskeletal Rearrangement, Glucose Transporter-4 Translocation, and Deoxyribonucleic Acid Synthesis* * This work was supported by Grants J 01287-Med and J 1584-Med from the Erwin Schrödinger Sti

P. Vollenweider, J. Olefsky, M. Czech, 1998, Endocrinology.

Effects of general receptor for phosphoinositides 1 on insulin and insulin-like growth factor I-induced cytoskeletal rearrangement, glucose transporter-4 translocation, and deoxyribonucleic acid synthesis.

J. Olefsky, J. Klarlund, M. Clodi, 1998, Endocrinology.

Persistent activation of phosphatidylinositol 3-kinase causes insulin resistance due to accelerated insulin-induced insulin receptor substrate-1 degradation in 3T3-L1 adipocytes.

J. Olefsky, H. Maegawa, A. Kashiwagi, 2000, Endocrinology.

Membrane-targeted Phosphatidylinositol 3-Kinase Mimics Insulin Actions and Induces a State of Cellular Insulin Resistance*

J. Olefsky, Prem M. Sharma, K. Egawa, 1999, The Journal of Biological Chemistry.

G Alpha-q/11 Protein Plays a Key Role in Insulin-Induced Glucose Transport in 3T3-L1 Adipocytes

John W. Adams, P. Vollenweider, J. Olefsky, 1999, Molecular and Cellular Biology.

The Functional Role of CrkII in Actin Cytoskeleton Organization and Mitogenesis*

J. Olefsky, A. Saltiel, D. Rose, 1999, The Journal of Biological Chemistry.

The Tumor Suppressor PTEN Negatively Regulates Insulin Signaling in 3T3-L1 Adipocytes*

J. Olefsky, R. Bookstein, T. Imamura, 2000, The Journal of Biological Chemistry.

G Alpha-q / 11 Protein Plays a Key Role in Insulin-Induced Glucose Transport in 3 T 3L 1 Adipocytes

John W. Adams, P. Vollenweider, J. Olefsky, 1999 .