Daphne van Leeuwen

发表

Novel Point Mutation in the Extracellular Domain of the Granulocyte Colony-Stimulating Factor (G-Csf) Receptor in a Case of Severe Congenital Neutropenia Hyporesponsive to G-Csf Treatment

M. Freedman, I. Touw, A. Ward, 1999, The Journal of experimental medicine.

EBV BILF1 Evolved To Downregulate Cell Surface Display of a Wide Range of HLA Class I Molecules through Their Cytoplasmic Tail

M. Ressing, F. Claas, E. Wiertz, 2013, The Journal of Immunology.

EBV Lytic-Phase Protein BGLF5 Contributes to TLR9 Downregulation during Productive Infection

M. Ressing, M. Buisson, E. Wiertz, 2011, The Journal of Immunology.

Varicelloviruses avoid T cell recognition by UL49.5-mediated inactivation of the transporter associated with antigen processing.

R. Tampé, J. Koch, M. Ressing, 2005, Proceedings of the National Academy of Sciences of the United States of America.

Human Cytomegalovirus-Encoded US2 Differentially Affects Surface Expression of MHC Class I Locus Products and Targets Membrane-Bound, but Not Soluble HLA-G1 for Degradation 1

M. Ressing, E. Wiertz, P. Le Bouteiller, 2003, The Journal of Immunology.

Specific Targeting of the EBV Lytic Phase Protein BNLF2a to the Transporter Associated with Antigen Processing Results in Impairment of HLA Class I-Restricted Antigen Presentation1

E. Kremmer, M. Ressing, N. Croft, 2009, The Journal of Immunology.

A CD8+ T cell immune evasion protein specific to Epstein-Barr virus and its close relatives in Old World primates

M. Ressing, J. Neefjes, N. Croft, 2007, The Journal of experimental medicine.

Host shutoff during productive Epstein–Barr virus infection is mediated by BGLF5 and may contribute to immune evasion

M. Ressing, M. Rowe, D. Ganem, 2007, Proceedings of the National Academy of Sciences.

Epstein-Barr Virus gp42 Is Posttranslationally Modified To Produce Soluble gp42 That Mediates HLA Class II Immune Evasion

T. Schumacher, M. Ressing, T. Ottenhoff, 2005, Journal of Virology.

Interference with T cell receptor–HLA-DR interactions by Epstein–Barr virus gp42 results in reduced T helper cell recognition

T. Schumacher, M. Schilham, M. Ressing, 2003, Proceedings of the National Academy of Sciences of the United States of America.

Amino acid composition of α1/α2 domains and cytoplasmic tail of MHC class I molecules determine their susceptibility to human cytomegalovirus US11‐mediated down‐regulation

E. Wiertz, P. Bouteiller, D. van Leeuwen, 2003, European journal of immunology.

Adenovirus-Specific CD4+ T Cell Clones Recognizing Endogenous Antigen Inhibit Viral Replication In Vitro through Cognate Interaction1

M. Schilham, M. Ressing, R. Toes, 2006, The Journal of Immunology.

Interaction Viral Replication in Vitro through Cognate Recognizing Endogenous Antigen Inhibit T Cell Clones + Adenovirus-specific Cd4

M. Schilham, M. Ressing, R. Toes, 2022 .

The DNase of Gammaherpesviruses Impairs Recognition by Virus-Specific CD8+ T Cells through an Additional Host Shutoff Function

M. Ressing, M. Rowe, Jaap M Middeldorp, 2007, Journal of Virology.

Rat cytomegalovirus induces a temporal downregulation of major histocompatibility complex class I cell surface expression.

D. Koppers-Lalic, G. Hassink, E. Wiertz, 2005, Viral immunology.

A CD8+ T cell immune evasion protein specific to Epstein-Barr virus and its close relatives in Old World primates

M. Ressing, J. Neefjes, N. Croft, 2007, The Journal of experimental medicine.

Signaling mechanisms coupled to tyrosines in the granulocyte colony-stimulating factor receptor orchestrate G-CSF-induced expansion of myeloid progenitor cells.

I. Touw, A. Ward, M. Hermans, 2003, Blood.