Manfred W. Baumstark

Homozygous familial defective apolipoprotein B-100. Enhanced removal of apolipoprotein E-containing VLDLs and decreased production of LDLs.

W. März, H. Scharnagl, M. Baumstark, 1997, Arteriosclerosis, thrombosis, and vascular biology.

Accumulation of "small dense" low density lipoproteins (LDL) in a homozygous patients with familial defective apolipoprotein B-100 results from heterogenous interaction of LDL subfractions with the LDL receptor.

W. März, H. Scharnagl, A. Berg, 1993, The Journal of clinical investigation.

Low density lipoprotein (LDL) subfractions during pregnancy: accumulation of buoyant LDL with advancing gestation.

W. März, M. Baumstark, H. Wieland, 2000, The Journal of clinical endocrinology and metabolism.

VLDL and IDL apolipoprotein B-100 kinetics in familial hypercholesterolemia due to impaired LDL receptor function or to defective apolipoprotein B-100.

M. Baumstark, R. Ninnis, A. Miserez, 1998, Journal of lipid research.