Mathew A. Vadas

发表

Three essential promoter elements mediate tumour necrosis factor and interleukin-1 activation of the granulocyte-colony stimulating factor gene.

M. F. Shannon, G. Goodall, M. Vadas, 1992, Growth factors.

A model for the interaction of the GM-CSF, IL-3 and IL-5 receptors with their ligands.

G. Goodall, M. Vadas, C. Bagley, 1993, Growth factors.

Monoclonal antibody 7G3 recognizes the N-terminal domain of the human interleukin-3 (IL-3) receptor alpha-chain and functions as a specific IL-3 receptor antagonist.

J. Gamble, G. Goodall, M. Vadas, 1996, Blood.

Recombinant human tumor necrosis factor-alpha. Regulation of N-formylmethionylleucylphenylalanine receptor affinity and function on human neutrophils.

M. Vadas, W. Marasco, A. Lopez, 1988, The Journal of clinical investigation.

Neutrophils activated by granulocyte-macrophage colony-stimulating factor express receptors for interleukin-3 which mediate class II expression.

M. Vadas, E. Korpelainen, C. Hawrylowicz, 1995, Blood.

Interleukin-5, interleukin-3, and granulocyte-macrophage colony-stimulating factor cross-compete for binding to cell surface receptors on human eosinophils.

M. Vadas, M. Elliott, D. Gillis, 1991, The Journal of biological chemistry.

Extracellular truncations of h beta c, the common signaling subunit for interleukin-3 (IL-3), granulocyte-macrophage colony-stimulating factor (GM-CSF), and IL-5, lead to ligand-independent activation.

G. Goodall, S. Barry, M. Vadas, 1996, Blood.

Cytokines Increase Human Hemopoietic Cell Adhesiveness by Activation of Very Late Antigen (VLA)-4 and VLA-5 Integrins By Jean-Pierre L6vesque,*~ David I. Leavesley,$ Silvana Niutta, g

Mathew A. Vadas, Paul J. Simmons, 1995 .