D. Erkelens

Abnormal postprandial apolipoprotein B-48 and triglyceride responses in normolipidemic women with greater than 70% stenotic coronary artery disease: a case-control study.

M. V. van Greevenbroek, T. D. de Bruin, D. Erkelens, 1996, Atherosclerosis.

Relationship between improved postprandial lipemia and low-density lipoprotein metabolism during treatment with tetrahydrolipstatin, a pancreatic lipase inhibitor.

T. D. de Bruin, M. Castro Cabezas, D. Erkelens, 1994, Metabolism: clinical and experimental.

Fasting and daylong triglycerides in obesity with and without type 2 diabetes.

M. Castro Cabezas, D. Erkelens, C. Halkes, 2003, Metabolism: clinical and experimental.

Menopause is associated with reduced protection from postprandial lipemia.

A. V. van Beek, T. D. de Bruin, D. Erkelens, 1999, Arteriosclerosis, thrombosis, and vascular biology.

Effect of intensive lipid-lowering strategy on low-density lipoprotein particle size in patients with type 2 diabetes mellitus.

A. Algra, G. Dallinga-Thie, J. Banga, 2001, Atherosclerosis.

Diurnal triglyceride profiles: a novel approach to study triglyceride changes.

M. Castro Cabezas, D. Erkelens, C. Halkes, 2001, Atherosclerosis.

Apolipoprotein Concentrations During Treatment and Recurrent Coronary Artery Disease Events

A. Zwinderman, E. E. van der Wall, D. Erkelens, 2000, Arteriosclerosis, thrombosis, and vascular biology.

Lipoprotein lipase-enhanced binding of lipoprotein(a) [Lp(a)] to heparan sulfate is improved by apolipoprotein E (apoE) saturation: secretion-capture process of apoE is a possible route for the catabolism of Lp(a).

T. D. de Bruin, H. van Barlingen, D. Erkelens, 1997, Metabolism: clinical and experimental.