Eran Leitersdorf

Frameshift and splice-junction mutations in the sterol 27-hydroxylase gene cause cerebrotendinous xanthomatosis in Jews or Moroccan origin.

V. Meiner, J. Cali, E. Leitersdorf, 1993, The Journal of clinical investigation.

The alpha-defensins stimulate proteoglycan-dependent catabolism of low-density lipoprotein by vascular cells: a new class of inflammatory apolipoprotein and a possible contributor to atherogenesis.

D. Rader, T. Ganz, K. Williams, 2000, Blood.

Evidence for a dominant gene that suppresses hypercholesterolemia in a family with defective low density lipoprotein receptors.

H. Hobbs, M. Brown, J. Goldstein, 1989, The Journal of clinical investigation.

Molecular genetics of familial hypercholesterolemia in Israel-revisited.

D. Harats, V. Meiner, R. Durst, 2017, Atherosclerosis.

Recent origin and spread of a common Lithuanian mutation, G197del LDLR, causing familial hypercholesterolemia: positive selection is not always necessary to account for disease incidence among Ashkenazi Jews.

Y. Friedlander, V. Meiner, F. Raal, 2001, American journal of human genetics.

Molecular genetics of familial hypercholesterolemia in Israel

R. Safadi, E. Leitersdorf, M. Gare, 1996, Human Genetics.

A new method for determination of serum cholestanol by high-performance liquid chromatography with ultraviolet detection.

D. Harats, E. Leitersdorf, M. Elisaf, 2000, Journal of chromatography. B, Biomedical sciences and applications.

Premature discontinuation of clinical trial for reasons not related to efficacy, safety, or feasibility.

G. Siest, P. Giral, E. Bruckert, 2001, BMJ : British Medical Journal.

Atherosclerosis in the apolipoprotein-E-deficient mouse: a decade of progress.

Eran Leitersdorf, E. Leitersdorf, K. Meir, 2004, Arteriosclerosis, thrombosis, and vascular biology.