E. Gelfand

发表

Jagged1 on Dendritic Cells and Notch on CD4+ T Cells Initiate Lung Allergic Responsiveness by Inducing IL-4 Production12

E. Gelfand, A. Joetham, J. Lucas, 2009, The Journal of Immunology.

Peroxisome Proliferator‐Activated Receptor‐g Agonist Treatment Increases Septation and Angiogenesis and Decreases Airway Hyperresponsiveness in a Model of Experimental Neonatal Chronic Lung Disease

David E. Irwin, M. Koster, E. Nozik-Grayck, 2009, Anatomical record.

Effect of Microbial Heat Shock Proteins on Airway Inflammation and Hyperresponsiveness1

E. Gelfand, A. Joetham, Y. Rha, 2002, The Journal of Immunology.

S-carboxymethylcysteine normalises airway responsiveness in sensitised and challenged mice

K. Takeda, N. Miyahara, T. Kodama, 2005, European Respiratory Journal.

Mast cell-derived tumour necrosis factor is essential for allergic airway disease

S. Reuter, C. Taube, R. Buhl, 2008, European Respiratory Journal.

Strain dependence of airway hyperresponsiveness reflects differences in eosinophil localization in the lung.

C. Irvin, K. Takeda, E. Gelfand, 2001, American journal of physiology. Lung cellular and molecular physiology.

IL-10 is necessary for the expression of airway hyperresponsiveness but not pulmonary inflammation after allergic sensitization.

M. Jordana, M. Mäkelä, E. Gelfand, 2000, Proceedings of the National Academy of Sciences of the United States of America.

Mice that overexpress Cu/Zn superoxide dismutase are resistant to allergen-induced changes in airway control.

A. Joetham, C. White, G. Larsen, 2000, American journal of physiology. Lung cellular and molecular physiology.

CD23 deficient mice develop allergic airway hyperresponsiveness following sensitization with ovalbumin.

E. Hamelmann, A. Joetham, H. Kikutani, 1997, American journal of respiratory and critical care medicine.

Anti-CD86 (B7.2) treatment abolishes allergic airway hyperresponsiveness in mice.

E. Hamelmann, G. Cieslewicz, A. Joetham, 1999, American journal of respiratory and critical care medicine.

Inhibition of IgE production and normalization of airways responsiveness by sensitized CD8 T cells in a mouse model of allergen-induced sensitization.

E. Gelfand, A. Kupfer, G. Lack, 1994, Journal of immunology.

Importance of myeloid dendritic cells in persistent airway disease after repeated allergen exposure.

E. Gelfand, A. Joetham, A. Dakhama, 2006, American journal of respiratory and critical care medicine.

Temporal association between airway hyperresponsiveness and airway eosinophilia in ovalbumin-sensitized mice.

E. Gelfand, G. Cieslewicz, C. Duez, 2001, American journal of respiratory and critical care medicine.

Leukotriene B4 receptor 1 is differentially expressed on peripheral T cells of steroid-sensitive and -resistant asthmatics.

E. R. Sutherland, Richard J Martin, E. Gelfand, 2014, Annals of allergy, asthma & immunology : official publication of the American College of Allergy, Asthma, & Immunology.

Critical roles for interleukin-4 and interleukin-5 during respiratory syncytial virus infection in the development of airway hyperresponsiveness after airway sensitization.

M. Mäkelä, E. Gelfand, G. Cieslewicz, 2000, American journal of respiratory and critical care medicine.

Reduction of antigen-induced airway hyperreactivity and eosinophilia in ICAM-1-deficient mice.

J. Drazen, A. Joetham, G. T. Sanctis, 1998, American journal of respiratory cell and molecular biology.

Dysregulation of metabolic pathways in a mouse model of allergic asthma

E. Gelfand, A. Joetham, M. Armstrong, 2017, Allergy. European Journal of Allergy and Clinical Immunology.

This information is current as Inflammation Airway Hyperresponsiveness and T Cells To Mediate + Alterations Poise CD 8 Stepwise Epigenetic and Phenotypic

A. Joetham, K. Takeda, Roland A. Marcus, 2013 .

in of leads to

G. Leikauf, K. Larson, K. Denzler, 2002 .

This information is current as Airway Disease The Role of IL-13 in Established Allergic

Y. Rha, A. Dakhama, C. Duez, 2002 .

Contrasting roles for the receptor for advanced glycation end-products on structural cells in allergic airway inflammation vs. airway hyperresponsiveness.

N. Miyahara, Y. Tanimoto, A. Kanehiro, 2015, American journal of physiology. Lung cellular and molecular physiology.

Allergic Airway Hyperresponsiveness-Enhancing γδ T Cells Develop in Normal Untreated Mice and Fail to Produce IL-4/13, Unlike Th2 and NKT Cells1

W. Born, C. Taube, N. Miyahara, 2009, The Journal of Immunology.

Inhibition of phosphodiesterase 4 attenuates airway hyperresponsiveness and airway inflammation in a model of secondary allergen challenge.

A. Joetham, A. Dakhama, A. Kanehiro, 2001, American journal of respiratory and critical care medicine.