Qian Li

GPR30 is necessary for estradiol-induced desensitization of 5-HT1A receptor signaling in the paraventricular nucleus of the rat hypothalamus

Q. Li, C. McAllister, N. Muma, 2012, Psychoneuroendocrinology.

Estradiol induces partial desensitization of serotonin 1A receptor signaling in the paraventricular nucleus of the hypothalamus and alters expression and interaction of RGSZ1 and Gαz

G. Carrasco, L. Kar, N. Muma, 2012, Neuropharmacology.

Evidence That Gz-Proteins Couple to Hypothalamic 5-HT1A Receptors In Vivo

L. D. van de Kar, G. Battaglia, F. García, 2000, The Journal of Neuroscience.

Chronic fluoxetine induces a gradual desensitization of 5-HT1A receptors: reductions in hypothalamic and midbrain Gi and G(o) proteins and in neuroendocrine responses to a 5-HT1A agonist.

L. D. van de Kar, Q. Li, L. Kar, 1996, The Journal of pharmacology and experimental therapeutics.

Daily injections of fluoxetine induce dose-dependent desensitization of hypothalamic 5-HT1A receptors: reductions in neuroendocrine responses to 8-OH-DPAT and in levels of Gz and Gi proteins.

L. D. van de Kar, G. Battaglia, F. García, 1999, The Journal of pharmacology and experimental therapeutics.

Fluoxetine gradually increases [125I]DOI-labelled 5-HT2A/2C receptors in the hypothalamus without changing the levels of Gq- and G11-proteins

G. Battaglia, Q. Li, L. Kar, 1997, Brain Research.